Can Clubfoot Come Back After Treatment? Relapse, Bracing Lapses and What Happens Next

Key Takeaways
- Clubfoot affects about 1 in 1,000 babies and involves both feet in roughly half of cases, according to the NHS.
- Relapse is most likely in the first few years after casting, which is exactly the window when night bracing is advised.
- Mayo Clinic and the NHS describe brace wear as about 23 hours a day for three months, then during sleep for several years, with the NHS citing an endpoint around age 4 to 5.
- Inconsistent or shortened brace wear is the strongest known modifiable predictor of clubfoot coming back.
- A dynamic relapse, where the foot is flexible but turns in during walking, is often addressed with casting followed by a tibialis anterior tendon transfer that redirects rather than removes the muscle.
- Heel blisters under the boots usually mean the heel is slipping and the fit is wrong; this is fixable and should be reported rather than tolerated.
Yes, clubfoot can come back after treatment. Relapse most often appears in the first few years of life, and the strongest known predictor is missed or shortened brace wear. A relapsed foot is usually treatable, often with a short new round of casting; some children later need a minor tendon procedure. The child's orthopedic team decides what is needed after examining the foot.
The boots-and-bar come off for the last time and there is a small celebration in the kitchen: a toddler in ordinary sandals, a parent who has counted twenty-three hours a day for years. Then, at a routine check some months later, the orthopedic specialist watches the child walk down the corridor twice, kneels, and gently tilts the heel. The foot is drifting. Nobody says the word straight away, but it hangs in the room: relapse.
Clubfoot relapse is one of the most searched and least explained parts of the whole clubfoot story. Families are told early that the brace matters, but rarely why a beautifully corrected foot would want to turn inward again, or what a return of the deformity actually means for a walking, running child.
This explainer takes that second conversation seriously. It covers what relapse is, why it happens, how much of it is about bracing lapses, what treatment usually follows, and which questions are worth asking before the next appointment.
What does clubfoot relapse actually mean?
Clubfoot, sometimes called talipes equinovarus, is a condition present at birth in which one or both feet point down and inward because the tendons and ligaments on the inner and back side of the foot are shorter and tighter than usual. According to the NHS it affects about 1 in every 1,000 babies, and in roughly half of those children both feet are involved.
Modern treatment for most babies is the Ponseti method: a series of gentle stretches and plaster casts, changed weekly, that bring the foot around over several weeks. Many children then have a tiny release of the Achilles tendon, called a tenotomy, followed by a brace of two boots joined by a bar. Mayo Clinic and the NHS both describe brace wear as full time for about three months and then during sleep for several years.
Relapse simply means that some part of the original deformity returns after the foot was corrected. In practice, clinicians describe it in degrees rather than as a switch that is either on or off. A foot may lose a little ability to bend upward at the ankle. The heel may start to tuck inward again. The forefoot may swing toward the midline, or the child may begin to bear weight on the outer edge of the foot. A full return to the original curled position is uncommon in a child who has been followed regularly, because the drift is usually noticed and addressed earlier.
Two further terms matter. A dynamic relapse is one in which the foot can still be moved passively into a good position but the child walks with it turned in, usually because of muscle imbalance. A rigid or structural relapse is one in which the foot has become stiff again and cannot be fully corrected by hand. Which of these a child has shapes everything that follows.
Why does a corrected clubfoot try to go back?
The honest answer is that the underlying tendency never fully disappears. Casting reshapes the position of the bones and stretches soft tissue, but it does not change the biology that produced the tight tissue in the first place. As Mayo Clinic notes, the exact cause of clubfoot is unknown, although family history, certain congenital conditions such as spina bifida, and low amniotic fluid in pregnancy are recognized risk factors.
Three forces work against a corrected foot during early childhood. The first is growth. Bones lengthen quickly in the first years of life, and the muscles and tendons on the inner side of the foot may not keep pace with the outer side, gradually pulling the foot back toward its old shape.
The second is muscle imbalance. The tibialis anterior is a muscle that runs down the front of the shin and lifts the foot; in many children with clubfoot it pulls more strongly toward the inside than the outside, so each step tugs the forefoot inward. Weaker outer-side muscles, the peroneals, cannot counterbalance it. Over thousands of steps a day, that small imbalance matters.
The third is scar and stiffness. Feet that have had extensive open surgery, which was the standard approach before casting methods became dominant, may be stiffer and behave differently when they relapse, which is why an older child’s history of operations is always relevant.
This is also why the brace exists. The boots hold the feet turned outward and the bar keeps them shoulder-width apart, so that while the child sleeps the tissues are stretched against the direction they would otherwise drift. The brace is not a treatment of the deformity; it is a nightly counterweight against recurrence during the years of fastest growth.
How common is clubfoot recurrence after Ponseti treatment?
Parents often want a single percentage, and it is worth explaining why no responsible clinician gives one with confidence. Published series define relapse in different ways, follow children for different lengths of time, and come from clinics with very different brace-wear patterns. A study that counts any loss of ankle flexibility will report a far higher figure than one that counts only feet needing new casts or surgery.
What the mainstream sources agree on is the direction of the evidence. Mayo Clinic states plainly that clubfoot may return if brace instructions are not followed, and that even with treatment the foot may not be entirely correctable. The NHS advises that there is a chance the condition can come back, and describes continued follow-up through childhood for that reason. Johns Hopkins Medicine describes relapse as a recognized part of the clubfoot pathway rather than a failure of the initial correction.
Three patterns recur across the literature summarized by these sources. Relapse is most likely in the first few years after casting, which is exactly the window during which night bracing is advised. Relapse is far more common in children whose brace wear was cut short or was inconsistent. And relapse becomes less likely with each year of growth completed in a good position, though it is not impossible in the older child.
Children whose clubfoot is part of a wider condition, such as arthrogryposis or a neuromuscular disorder, tend to have stiffer feet and higher relapse rates than children with isolated, or idiopathic, clubfoot. Their treatment plans are usually set up with that expectation from the start.
The practical message is not a number. It is that every child treated for clubfoot should be seen regularly by an orthopedic team until skeletal growth is well advanced, because catching drift early keeps treatment small.
Signs of clubfoot relapse: what clinicians look for and what parents tend to notice first
Relapse is diagnosed in the clinic, by examination, and no description here replaces that. It still helps families to understand what the specialist is checking, because parents are usually the first to see change simply because they watch the child walk every day.
The examiner will typically look at the resting shape of the foot when the child stands, then at the heel from behind to see whether it sits straight or tilts inward. The ankle is bent upward with the knee straight to measure how far the foot can flex, and the same movement is compared with the other side. The forefoot is checked to see whether it points straight ahead or curves toward the midline. The child is then asked to walk, and often to run, barefoot along a corridor.
What parents commonly describe when relapse is later confirmed includes shoes wearing unevenly along the outer edge, a child who seems to trip more than peers, a foot that appears to turn in during walking even though it looks straight at rest, and difficulty with the heel touching the floor comfortably. None of these observations proves relapse; toddlers walk unevenly for many reasons, and in-toeing is common in young children without any clubfoot history. Raising them at the next visit, or bringing the visit forward, is the right response.
Some centers add formal gait analysis, in which cameras and pressure plates record how the foot moves and where weight lands during walking. This can pick up subtle patterns, such as reduced upward bend at the ankle during the swing of the leg, before they are obvious to the eye. It is a research and planning tool rather than something every child needs.
X-rays are used selectively, mainly in older children or when surgery is being considered, because much of the young foot is still cartilage and does not show well on film.
Clubfoot brace not worn: why bracing lapses matter more than anything else
If one message deserves the whole page, it is this: the single modifiable factor in clubfoot relapse is brace wear. Mayo Clinic and the NHS describe the same schedule in slightly different words, roughly 23 hours a day for the first three months after casting, then at night and nap time. The NHS gives the typical endpoint as around 4 to 5 years of age; Mayo describes night wear lasting up to about three years. The child’s own team sets the end date.
Why lapses are so consequential comes back to mechanism. The brace works by holding a stretch during the hours of fastest tissue growth. Miss a night here and there and the tissue simply loses a little of that counterbalance. Miss weeks, especially in the first year after casting, and the muscles on the inner side of the foot are free to shorten as the bones lengthen, which is precisely the drift the brace was preventing.
Lapses rarely happen out of carelessness. Common reasons reported to clinicians include a child who cries when the boots go on, red marks or blisters on the heel, boots that have become too small, a period of illness or travel, a change in who is doing bedtime, or the reasonable but mistaken belief that a foot that looks perfect no longer needs the brace. Each of these has a practical answer, and the team would far rather hear about a struggle than discover a relapsed foot at a later visit.
Skin problems deserve particular attention. Heel blisters usually mean the heel has slipped up inside the boot, often because the strap over the ankle is not snug or the boot is the wrong size. This is fixable, and persistent skin breakdown should never be tolerated in silence.
Children who have relapsed and been re-corrected are almost always placed back in the brace afterward, sometimes for longer than originally planned. Understanding the reason, rather than experiencing it as a punishment, tends to make the second period of bracing go more smoothly.
How clubfoot relapse is treated: what actually happens
Treatment of a relapsed foot follows the same logic as the original correction, scaled to how far the foot has drifted and how stiff it has become.
For most young children with a mild to moderate relapse, the first step is a return to casting. Weekly plaster casts, usually fewer than were needed at birth, gradually bring the foot back around. Mayo Clinic describes this repeat casting as a standard response when the foot begins to recur. If the Achilles tendon has tightened again, a repeat tenotomy may be discussed; this is a small procedure in which the tendon at the back of the heel is released so the ankle can bend upward, and it heals over a few weeks in a cast.
Once the foot is back in position, bracing restarts. For a young child this typically means a further period of night wear, and the team may extend the total bracing period beyond what was first planned.
When the relapse is dynamic, meaning the foot can be corrected by hand but turns in during walking because the tibialis anterior pulls too strongly inward, the foot is often first restored with casting and then a tendon transfer is considered. This is described in detail in the next section.
For a rigid relapse in an older child, or a foot that has had previous open surgery and is scarred, the options broaden and become more individual. They may include longer casting, release of tight soft tissue, or procedures on bone if the shape has become fixed. These are decisions made after careful examination and, usually, imaging, and they sit entirely with the treating team.
Physiotherapy is frequently woven through all of this, focusing on stretching the calf and inner foot and strengthening the outer muscles of the lower leg. It supports the other treatments; it is not a substitute for them.
Clubfoot relapse surgery: the tibialis anterior tendon transfer explained
The operation most often associated with clubfoot relapse is the tibialis anterior tendon transfer. Because it appears in search results with intimidating titles, it helps to describe it plainly.
The tibialis anterior is the muscle you can feel tensing at the front of your shin when you lift your foot. Its tendon normally attaches toward the inner side of the midfoot. In a child whose foot turns in during walking, that attachment point means every step pulls the foot inward. The transfer moves the tendon’s attachment to a bone in the middle or outer part of the foot, so the same muscle now lifts the foot straight up rather than up and in. The muscle is not removed or weakened; it is redirected.
The procedure is usually considered only after the foot has been brought back into a good passive position with casting, because a tendon cannot pull a stiff foot straight. It is typically offered to children old enough that the bone the tendon is fixed to has developed enough to hold it, which is why it is a treatment for the walking child rather than the infant.
Afterward the leg is placed in a cast, commonly for around six weeks, while the transferred tendon heals into its new position. Bracing after the operation varies between teams; some children do not need to return to night boots because the muscle balance itself now resists the inward drift, which is one of the reasons the procedure is valued.
Risks are those of any operation in a child: infection, wound problems, the tendon stretching or pulling away from its new attachment, and, in a minority, either too little correction or an over-correction that turns the foot outward. Anesthesia is administered by a pediatric anesthesia team and discussed with the family beforehand.
Whether a tendon transfer is appropriate, and when, is a judgment made by the surgeon after examining the child walking and checking the foot’s flexibility. It is one option among several, not an automatic next step.
Who is usually treated right away for clubfoot relapse, and who is asked to wait?
Not every change in a treated foot is acted on immediately, and families are sometimes unsettled by a specialist who chooses to watch rather than treat. Understanding the reasoning helps.
Children usually offered prompt treatment include those with a clear loss of upward ankle bend, particularly if the heel is no longer reaching the floor comfortably in standing; those whose heel has begun tilting inward again; those with a rigid element that cannot be corrected by hand; and young children in whom a short course of casting is likely to restore position quickly while the tissues are still very pliable. In these cases waiting tends to mean more casts later rather than fewer.
Children who are often asked to wait and be reviewed include those with mild in-toeing during walking but a fully flexible foot, since many toddlers in-toe for reasons unrelated to clubfoot and the pattern may settle; those whose brace fit has just been corrected after a period of poor wear, to see whether consistent bracing alone holds the foot; and children nearing the age at which a tendon transfer could be performed, where a few months of observation allows one definitive procedure rather than two staged ones.
A further group is children with clubfoot linked to a wider neuromuscular or syndromic condition. Their feet may relapse more readily and respond less predictably, and their orthopedic care is usually planned alongside neurology or genetics teams. Timelines for these children are set individually.
Age changes the calculation in both directions. Very young children are re-cast readily because their tissues respond well. Older children with stiff feet may be advised to wait for growth to slow before a bony procedure, or conversely to act before a fixed deformity becomes harder to address. None of these decisions can be made from a description; they follow examination by the treating team.
Clubfoot relapse at a glance: types, typical responses and what parents can expect
The table below summarizes how clinicians commonly group relapse and the responses most often discussed. It is a map of typical pathways, not a plan for any individual child; the team may reasonably choose a different route.
| Type of relapse | What the examiner finds | Response most often discussed | Typical timeline (per NHS and Mayo Clinic descriptions) |
|---|---|---|---|
| Early drift, flexible | Slight loss of ankle bend or heel tilt; foot fully correctable by hand | Check brace fit and wear; short course of weekly casts; resume night bracing | A few weekly casts; bracing continued to around 4–5 years of age |
| Tight Achilles | Ankle will not bend up past neutral with knee straight | Casting, often with repeat tenotomy; return to bracing | Cast for several weeks after tenotomy while tendon heals |
| Dynamic in-toeing | Foot straight at rest but swings inward during walking; flexible | Casting to restore position, then tibialis anterior tendon transfer in the walking child | Roughly six weeks in cast after transfer; bracing afterward varies by team |
| Rigid or structural | Foot stiff, not fully correctable; often older child or prior open surgery | Individual plan: longer casting, soft tissue release, or bony procedures | Set case by case after imaging and examination |
Two features of the table deserve comment. First, the response to every type begins with a look at bracing, because a foot that has drifted for want of brace wear may be held by consistent wear once re-corrected. Second, timelines are given as ranges drawn from general descriptions in the cited sources, and individual children move through them faster or slower depending on how the foot responds and how the child tolerates casts.
The table also makes visible why regular follow-up is protective: the top row is a far smaller undertaking than the bottom one, and children who are seen every few months in early childhood tend to be treated in the top two rows.
What the following weeks usually look like after relapse treatment
Families who have been through casting once usually find the second time more predictable, though a toddler is a different patient from a newborn.
During repeat casting, the leg is enclosed in plaster from toes to thigh and the cast is changed roughly weekly. Children of walking age are generally kept off the cast or allowed only limited weight through it, depending on the team’s instructions, and this can be the hardest part for an active child. Parents are asked to watch that toes stay pink and warm and that the cast does not slip or become soft. Skin care, distraction and keeping the cast dry occupy most of the week between visits.
After a tenotomy, the foot is casted with the ankle bent up and held for several weeks while the tendon heals, as described by the NHS. Discomfort is usually brief and managed with the comfort measures the team recommends; medicine choices, if any, are the prescribing clinician’s to make.
After a tendon transfer, the child wakes with the leg in a cast and typically goes home the same day or after one night. Around six weeks later the cast comes off, the foot is examined, and the child begins walking again, sometimes with a period of physiotherapy to rebuild confidence and strength. Stiffness for a few weeks after cast removal is expected and eases with use.
Bracing then resumes for most children who have been re-cast, and the team will explain how long. It helps to check the fit of the boots carefully at this point, since feet have often grown during treatment and an ill-fitting boot is a common reason for renewed lapses.
Follow-up visits are usually more frequent for the year after a relapse and then spread out again. Growth continues to be checked until the foot is skeletally mature.
Can clubfoot go back to normal, and is a treated clubfoot a disability?
These two questions arrive together in almost every consultation, and they deserve a direct, honest answer.
A treated clubfoot is not identical to a foot that was never affected. Mayo Clinic notes that even with treatment the foot may not be completely correctable. Commonly, the treated foot is slightly shorter and a little wider than the other, the calf on that side is thinner, and there may be somewhat less upward bend at the ankle. These differences are usually small and rarely limit activity. MedlinePlus describes the outcome after early treatment as usually good, with most children walking without a limp.
What matters to families is function. Children treated early, and followed for relapse, generally walk, run, play sport and wear ordinary shoes, sometimes with a slight size difference between feet. Some adults describe stiffness or aching after heavy activity, and a minority develop arthritis in the midfoot later in life, more often after extensive childhood surgery than after casting-based treatment.
Whether clubfoot counts as a disability depends on the definition being used. In a medical and everyday sense, a well-treated foot with good function is not disabling. In legal or educational terms, definitions vary by country and by the individual’s actual limitations, and a child with a stiff, painful or repeatedly relapsed foot might qualify for support that a child with a flexible, painless foot would not. Families are best served by asking the treating team to document function precisely if such a question arises.
The phrase “back to normal” is perhaps the wrong target. A more useful one is a foot that is plantigrade, meaning flat on the ground, flexible, painless and strong enough for whatever the child wants to do. That is what treatment, and vigilance against relapse, is trying to protect.
What people often get wrong about clubfoot relapse
Myths gather around any long treatment, and several deserve gentle correction.
“The foot is fixed once the casts come off.” Casting achieves correction; bracing maintains it. Mayo Clinic is explicit that clubfoot may return if the brace is not worn as instructed. The finish line is the end of bracing and growth, not the last cast.
“Relapse means the first treatment failed.” The tendency to relapse is built into the condition, not into the quality of the initial correction. Feet that were beautifully corrected relapse when growth outpaces the soft tissue, which is why follow-up is planned rather than optional.
“Once a child is walking, the brace is pointless.” The reverse is closer to the truth: the early walking years are when relapse is most likely, and the NHS describes night bracing continuing until around 4 to 5 years of age for that reason.
“Clubfoot means a high-risk pregnancy.” Isolated clubfoot seen on a prenatal scan does not itself make a pregnancy high risk. It prompts a careful look for other findings and a conversation with the maternity team, and most such babies are otherwise healthy. Mayo Clinic lists factors such as family history and low amniotic fluid as associations, not consequences.
“Clubfoot is linked to autism.” No mainstream evidence supports a causal link between isolated clubfoot and autism. Clubfoot can occur as part of some genetic syndromes that also affect development, which is why a child with additional features is assessed more broadly, but for the great majority of children with isolated clubfoot there is no connection.
“Relapse can be fixed at any age just as easily.” A flexible relapse in a two-year-old and a rigid one in a nine-year-old are different problems, and the later one is harder. This is the strongest argument for keeping every follow-up appointment even when the foot looks fine.
Questions to ask your care team about clubfoot recurrence
Consultations are short and emotions run high, so it helps to walk in with questions written down. These are the ones experienced clinicians say they most wish families would ask.
- Is this a dynamic relapse or a structural one, and how does that change the plan?
- How much upward ankle movement does my child have on each side, and what would you like it to be?
- Do you think brace wear contributed here, and if so, what specifically can we change about fit, straps or routine?
- Are the boots the correct size now, and how often should they be checked as feet grow?
- If casting is planned, roughly how many casts do you expect, and can my child put weight on the cast?
- Will a repeat tenotomy be needed, and how will we know?
- Is a tendon transfer being considered now or later, and what would make you decide either way?
- How long will bracing continue after re-correction, and what is the target end point?
- Would gait analysis or X-rays add anything to the decision in my child’s case?
- What should I watch for at home between visits, and how quickly should I contact you if I see it?
- How often will follow-up be for the next year, and until what age will my child be seen?
- Is there anything about my child’s wider health or family history that changes the likelihood of another relapse?
Two further habits pay off. Ask the team to demonstrate how they check the foot, so that home observations are grounded in what the clinic actually measures. And ask for the plan in writing, including the bracing schedule, because bedtime routines pass between parents, grandparents and carers, and consistency is the whole point.
Every one of these decisions rests with the treating orthopedic team, who can examine the foot, watch the child move and weigh the individual history. The value of good questions is not to second-guess that judgment but to understand it well enough to carry it out at home.
When to call your doctor
Most changes in a treated clubfoot are gradual and belong at the next scheduled visit, brought forward if a parent is concerned. A smaller set of situations warrant a call the same day.
During casting or after any procedure, contact the team promptly if the toes become pale, blue, cold or very swollen, if the child cannot move the toes or complains of pins and needles, if there is persistent pain not eased by repositioning and the comfort measures advised, or if the cast slips, cracks, becomes soaked or develops a strong odor or discharge. A fever after tenotomy or tendon transfer, or redness and spreading warmth around a wound, also needs same-day advice.
Around bracing, seek advice within a day or two if the skin under the boots blisters, breaks or bleeds, or if the child develops red marks that do not fade within an hour of removing the boots. Skin problems are almost always a fit issue and are fixable; they should not lead to quietly abandoning the brace.
Between visits, ask for an earlier appointment if the foot appears to be turning in again at rest, if the heel no longer sits flat when the child stands, if the child starts walking on the outer edge of the foot, tripping markedly more than before, or limping, or if a previously flexible ankle seems stiff. These are signals for assessment, not diagnoses.
Seek urgent care through the usual emergency channels for any child with a hot, swollen limb accompanied by high fever or drowsiness, or for a limb that has lost color or sensation, regardless of clubfoot history.
When in doubt, the team would rather hear from you. An unnecessary call costs a few minutes; an unreported drift can cost months of extra casting.
Frequently asked questions
Can clubfoot go back to normal after treatment?
A treated clubfoot usually works well but is rarely identical to an unaffected foot. The foot is often slightly smaller, the calf thinner and ankle flexibility a little reduced. MedlinePlus describes the outcome after early treatment as usually good, with most children walking without a limp, running and wearing ordinary shoes. Function, not perfect symmetry, is the realistic goal.
What are the first signs of clubfoot relapse?
Clinicians look for loss of upward ankle bend, a heel that tilts inward, a forefoot curving toward the midline, or weight bearing on the outer edge of the foot during walking. Parents often notice uneven shoe wear or more tripping first. None of these confirms relapse; they are reasons to bring the next orthopedic visit forward for examination.
What happens if the clubfoot brace is not worn?
The brace holds a stretch during the years of fastest growth, so missed wear lets the inner-side tissues shorten as bones lengthen. Mayo Clinic states clubfoot may return if instructions are not followed. If lapses have happened, the team checks the foot, corrects the boot fit and may recommend a short course of casting before resuming bracing.
How common is clubfoot recurrence after Ponseti treatment?
No single reliable percentage exists, because studies define relapse differently and brace-wear patterns vary widely between centers. Mainstream sources including Mayo Clinic, the NHS and Johns Hopkins Medicine describe relapse as a recognized possibility, most likely in early childhood and strongly linked to inconsistent bracing, which is why follow-up continues for years after casting ends.
What does clubfoot relapse surgery involve?
The procedure most often discussed is the tibialis anterior tendon transfer, which moves the attachment of a shin muscle from the inner to the middle or outer foot so it lifts the foot straight rather than inward. It is used for flexible relapses in walking-age children after casting, followed by roughly six weeks in a cast. The surgeon decides whether and when it is appropriate.
Is there a link between clubfoot and autism?
No mainstream evidence shows that isolated clubfoot causes or is caused by autism. Clubfoot can appear as one feature of certain genetic syndromes that also affect development, so a child with additional findings is assessed more broadly. For the large majority of children whose clubfoot occurs on its own, there is no established connection.
Is clubfoot a high-risk pregnancy?
Isolated clubfoot seen on a prenatal scan does not by itself make a pregnancy high risk. It usually prompts a detailed scan to look for other findings and a conversation with the maternity team about the treatment pathway after birth. Mayo Clinic lists family history and low amniotic fluid as associations, and most affected babies are otherwise healthy.
Is having a clubfoot considered a disability?
In a medical sense, a well-treated clubfoot with good function is not disabling; most children run, play sport and wear ordinary shoes. Legal or educational definitions vary by country and depend on actual limitation, so a stiff, painful or repeatedly relapsed foot might qualify for support where a flexible, painless one would not. The treating team can document function if needed.
How long does a child wear the brace after a clubfoot relapse is treated?
Most children who are re-cast return to night bracing afterward, and the team may extend the period beyond the original plan. The NHS describes routine night bracing continuing until around 4 to 5 years of age. Children who have a tendon transfer may or may not need to resume the brace, depending on their team’s approach and the foot’s response.
Can clubfoot relapse in an older child or teenager?
It can, though it is less common once several years of growth have passed with the foot in a good position. Relapse in older children is more often rigid and may involve prior surgery or scarring, so the treatment plan is more individual and may include soft tissue or bony procedures. Regular orthopedic review until skeletal maturity is the safeguard.
References
This article is for general information only and is not a substitute for professional medical advice. Please consult a qualified doctor about your individual situation.
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