Is Kawasaki Disease Contagious? Myths About Causes, Spread and Who Gets It, Corrected

It is a little after two in the morning on a pediatric ward. A toddler is finally asleep, his lips still cracked and red, a hospital bracelet loose on a swollen wrist. His mother is not asleep. She has a question she has already asked two nurses and is now typing into her phone: is Kawasaki disease contagious, and should her daughter stay away from preschool tomorrow?

It is the most natural question in the world. The illness looks infectious. It arrives with a high fever, a rash, red eyes and a name most parents have never heard until a doctor says it out loud. Everything about it resembles the childhood infections that sweep through a daycare in a week.

And yet the answer is a firm no. Understanding why doctors can be so certain about that, while still admitting they do not know what causes it, is the best way to make sense of the whole illness, from the first day of fever to the last follow-up scan.

Is Kawasaki disease contagious? What the evidence actually shows

Every major health body that describes this illness says the same thing: it does not spread between people. The CDC, the NHS and Mayo Clinic all state plainly that Kawasaki disease is not contagious, and that a child with it poses no infection risk to siblings, classmates or the adults caring for them.

That confidence rests on decades of looking. If a virus or bacterium were passing from child to child, researchers would expect to find it in blood, throat swabs or tissue samples, and they would expect the illness to move through households and classrooms the way chickenpox or strep throat does. Neither happens. Second cases within a family are uncommon, and when they do occur they are explained far better by shared genes than by shared germs. Pediatric nurses and doctors who treat dozens of these children over a career do not develop the illness themselves.

What makes the question reasonable is that the disease behaves oddly for something noncontagious. Cases in the United States rise in winter and early spring, according to the American Heart Association’s scientific statement on the condition. Japan has recorded nationwide waves of cases. Those patterns look like the footprint of something circulating in the community.

The most widely held explanation resolves that tension. Something common in the environment, probably a germ most children meet without any drama, appears to set off an exaggerated immune reaction in a small number of children who are genetically primed for it. The trigger may travel; the disease does not. Pollen drifts across a whole town, but only some people get hay fever, and nobody catches hay fever from a sneezing neighbor. The illness is the child’s own immune response, and an immune response cannot be transmitted.

In practical terms, no isolation is needed, a sibling can go to school, and a parent can hold, kiss and comfort the child freely.

What causes Kawasaki disease, if not a germ you can catch?

Honesty first: nobody knows. A Japanese pediatrician first described the illness decades ago, and despite an enormous research effort since, no single cause has been confirmed. What exists instead is a set of strong clues that point in the same direction.

The age pattern is the first clue. The illness is rare in babies under six months, who still carry antibodies passed from their mothers, and rare after early childhood, by which time most children have met the common germs of the nursery. The peak sits in between, in the window when a child’s immune system is meeting the world largely on its own. That is exactly the pattern you would expect from an infectious trigger.

Genetics is the second clue. Children of Japanese and other East Asian ancestry have higher rates wherever in the world they live, which argues for inherited susceptibility rather than local environment alone. The AHA statement notes that in Japan a sibling of an affected child carries roughly a tenfold higher risk than the general population, and that children whose parents had the illness are also at increased risk. Researchers have linked several genes involved in immune signaling to susceptibility, though none is decisive on its own.

The third clue is the seasonal clustering already mentioned. Candidate triggers studied over the years include ordinary respiratory viruses, bacterial toxins that overstimulate immune cells, and even airborne particles carried on wind currents from Central Asia. Each remains a hypothesis. None has been proven, and this article will not pretend otherwise.

What the evidence does rule out matters just as much. There is no established link to diet, breastfeeding choices, household cleaning products, pets or routine childhood vaccines. It is not caused by anything a parent did or failed to do.

How many kids get Kawasaki disease, and who is most likely?

The numbers are small in absolute terms and large in significance. In the United States, the AHA scientific statement puts the annual incidence at roughly 20 cases per 100,000 children under five. In Japan the same statement records rates above 250 per 100,000 in that age group, more than ten times higher. Korea and Taiwan also report high rates. Most other countries fall somewhere near the US figure.

Put another way, a typical American preschool classroom will almost never see a case, and a general pediatrician may encounter only a handful across a working life. Yet because the illness can damage the coronary arteries, the vessels that feed the heart muscle, Mayo Clinic and the AHA both describe it as the leading cause of acquired heart disease in children in developed countries. Acquired here simply means heart disease a child was not born with.

Who is most likely to be affected follows a consistent pattern across studies cited in the AHA statement:

• Age: about three-quarters of cases occur in children under five, with a peak between one and two years.
• Sex: boys are affected roughly one and a half times as often as girls.
• Ancestry: children of East Asian, and especially Japanese, descent carry the highest risk, but children of every background can develop it.
• Family history: a sibling or parent who had the illness raises the odds, though the absolute risk to that sibling remains low.

None of these factors is something a family can change, and none makes diagnosis automatic. A girl of European ancestry with a five-day fever deserves the same careful look as a boy who fits the textbook profile. Doctors are trained to hold the statistics lightly and examine the child in front of them.

Kawasaki disease in adults: can grown-ups get it?

Cases in adults have been reported in the medical literature, but they are genuinely rare, and most people who search for Kawasaki disease in adults are asking one of two quite different questions.

The first is whether an adult with a prolonged fever, rash and red eyes might have it. Occasionally, yes, and case reports describe young adults with the classic picture. Far more often, though, an adult with those features turns out to have something else that overlaps: a severe drug reaction, a toxin-mediated bacterial illness, a viral infection, or an inflammatory syndrome that can follow a recent infection. Because there is no single confirming test, doctors in these situations work by excluding the alternatives first. The AHA statement discusses this overlap and stresses that adult diagnosis is unusual and requires specialist judgment.

The second question comes from adults who had the illness as children and want to know what it means for them now. Here the answer is more concrete. Whether follow-up is needed depends almost entirely on what the heart scans showed in the weeks after the original illness. The AHA recommends lifelong cardiology follow-up for people whose coronary arteries were changed, with the intensity matched to the size and course of those changes. People whose scans stayed normal are generally considered to have a low long-term risk from the illness itself, although the statement encourages ordinary attention to heart health as they age, since the long-term picture is still being studied.

Adults who cannot find their childhood records but remember the illness can reasonably mention it to a primary care clinician. Whether anything more is needed is a decision for that clinician, based on history and examination, not something to settle from an article.

What actually happens in the body during Kawasaki disease

The medical term is vasculitis, which means inflammation of the walls of blood vessels. That single sentence explains almost everything visible and invisible about the illness.

Picture an artery as a flexible hose with three layers: a smooth inner lining, an elastic and muscular middle layer, and a tough outer coat. In this illness, for reasons still unclear, immune cells swarm into the middle layer of medium-sized arteries around the body. The inflamed walls swell and leak. Where those arteries run close to the skin or the mucous membranes, the result is what a parent sees: flushed cheeks, red bloodshot eyes without sticky discharge, cracked lips, a tongue that looks like a strawberry, puffy hands and feet, and swollen glands in the neck.

The arteries no one can see are the ones that matter most. The coronary arteries, which wrap around the heart and supply its muscle with blood, are about the diameter of a drinking straw in a small child. When their elastic layer is inflamed and weakened, the wall can stretch outward under the pressure of each heartbeat, forming a bulge called an aneurysm. Most of that stretching happens in the second and third weeks, often after the fever has already settled, which is why follow-up scans are scheduled even in a child who looks well.

One more process runs in the background. Around the second week, the bone marrow releases a surge of platelets, the cell fragments that plug leaks and start clots. Cleveland Clinic and Mayo Clinic both describe this rise as a typical feature. Inside a healthy artery it causes no trouble. Inside a roughened, ballooned artery, extra platelets raise the chance of a clot forming where it can block blood flow.

Every part of the treatment plan is aimed at one of those two problems: cooling the inflammation before the wall is damaged, and keeping blood from clotting where damage has already occurred.

Kawasaki disease symptoms timeline: when does the rash appear, and what follows?

Doctors describe the illness in three overlapping phases. The timings below are typical ranges drawn from the NHS and AHA descriptions, not a schedule any individual child will follow exactly, and they are offered to help families understand what the care team is watching for rather than to diagnose at home.

The acute phase covers roughly the first one to two weeks. Fever comes first, usually high and stubbornly unresponsive to the usual measures. The AHA statement notes that without treatment the fever lasts an average of about eleven days and can run for three to four weeks. The rash usually appears within the first five days of fever, according to both the AHA and Mayo Clinic. It varies enormously: blotchy red patches on the trunk, a fine measles-like spread, or redness concentrated in the diaper area. Red eyes, cracked lips, a strawberry tongue, swollen hands and feet, and an enlarged gland on one side of the neck tend to cluster in this same window. Irritability is striking; parents often say their child seems miserable out of all proportion to what they can see.

The subacute phase runs from roughly week two to week four. The fever and rash fade, but the skin on the fingertips and toes begins to peel in sheets, a change Mayo Clinic places in the second to third week. Joint aches are common. Platelet counts peak. This is also the period when coronary aneurysms are most likely to form, which is why it is the phase doctors worry about most despite the child looking better.

The convalescent phase, from about week four to week six and sometimes longer, is a slow return to normal. Blood markers of inflammation settle. Horizontal grooves may appear across the fingernails one to two months after the illness, a harmless echo of the fever that the AHA describes as a late sign. Energy returns gradually, and many children are noticeably tired for several weeks.

How doctors diagnose Kawasaki disease when there is no single test

There is no blood test, swab or scan that says yes or no. Diagnosis is a judgment built from the pattern of the illness over days, supported by laboratory results and an ultrasound of the heart. That is uncomfortable for parents who want certainty, but it is the honest state of the science.

The AHA criteria, used worldwide, ask for fever lasting at least five days together with at least four of five principal features: the rash, the red eyes, the changes in the lips and mouth, the changes in the hands and feet, and the swollen neck gland. When a child has the fever plus only two or three features, doctors may consider what is called incomplete Kawasaki disease. Incomplete does not mean mild. It means the picture is harder to read, and the AHA provides a separate pathway using blood results and heart ultrasound to decide whether treatment is warranted.

Blood tests look for the fingerprints of widespread inflammation: raised inflammatory markers, a high white cell count, anemia, low protein levels, mildly disturbed liver tests and, from the second week, a climbing platelet count. A urine sample often shows white cells without bacteria. None of these results is unique to this illness, but together they build a case.

The echocardiogram, an ultrasound scan of the heart done with a probe on the chest, is the closest thing to a specific test. Bright, thickened or widened coronary arteries at the time of diagnosis strongly support the diagnosis and change the urgency of treatment. A normal early scan does not rule the illness out, because artery changes usually appear later.

Doctors will also test for the infections the illness resembles. A positive throat swab for strep, for example, does not automatically exclude the diagnosis, a point the AHA statement makes explicitly, because children can carry strep and have this illness at the same time.

How Kawasaki disease is treated: what the drip and the tablets are doing

Treatment has two goals that map onto the two problems described earlier: switch off the inflammation quickly, and reduce the chance of clots while the arteries recover. The medicines involved are described here by mechanism and typical timeline only. Every decision about which medicine, how much and for how long rests with the treating team.

The cornerstone is intravenous immunoglobulin, usually shortened to IVIG. It is a purified pool of antibodies collected from many thousands of blood donors and given through a vein as a slow infusion. The AHA statement describes a single infusion given over roughly ten to twelve hours, and recommends starting it within the first ten days of fever when possible, because that is the window in which it most clearly reduces coronary damage. It is still given after day ten if inflammation is ongoing. Exactly how IVIG calms the immune storm is not fully understood; it appears to act on several arms of the immune system at once, damping the signals that keep immune cells attacking vessel walls. In most children the fever falls away within a day or two of the infusion.

Aspirin is the second component, and it puzzles many parents who have been told never to give aspirin to a child. The NHS explains the apparent contradiction: aspirin is normally avoided in children under sixteen because of a rare liver and brain complication, but this illness is one of the recognized exceptions, used under specialist direction. In the early phase it is used for its anti-inflammatory effect. Once the fever has settled, it is continued at a different level for its antiplatelet effect, making platelets less sticky while their numbers are high. How long it continues depends on what the follow-up heart scans show.

The AHA statement reports that in roughly 10 to 20 percent of children the fever persists or returns after IVIG. Options in that situation include a second infusion, corticosteroids, or other immune-modulating medicines, chosen by the team according to the child’s picture.

Who is treated right away, and who is asked to wait and be watched

Because the diagnosis is a judgment, so is the decision about when to start treatment. Families sometimes experience the waiting as hesitation. It is usually the opposite: a deliberate use of time to let the pattern declare itself.

Children treated promptly include those who meet the full criteria after five days of fever, those with an incomplete picture whose blood results and heart scan point clearly toward the diagnosis, and any child in whom the coronary arteries already look abnormal. The AHA also singles out infants under six months with a prolonged unexplained fever for a low threshold to investigate and treat, because this group has a higher risk of artery involvement and often shows fewer of the classic outward features.

Children who may be asked to wait, typically with a repeat examination and repeat blood tests within a day or two, include those whose fever has not yet lasted five days and who have a plausible alternative explanation such as a confirmed viral infection, and those whose inflammatory markers are low and whose features are fading on their own. Waiting is done with close follow-up, not dismissal, and the door back to the emergency department is always open.

There is a specific reason the five-day mark carries weight. The AHA statement notes that giving IVIG before the fifth day of fever has not been shown to produce better outcomes and may be associated with a greater need for repeat treatment. Treating earlier is not automatically safer, which runs against intuition and is worth hearing from the team directly.

Alternatives and risks are part of the same conversation. IVIG is a blood product, carries a small risk of infusion reactions and, rarely, of more serious reactions; aspirin carries bleeding and other risks; corticosteroids have their own profile. A good team will lay these out plainly, and the decision remains theirs to make with the family, never a matter of protocol alone.

Kawasaki disease versus the illnesses it is mistaken for

Much of the confusion about contagiousness comes from the company this illness keeps. Several genuinely infectious childhood illnesses produce fever, rash and red eyes, and doctors have to weigh each one. The table sets them side by side, drawing on the descriptions in the AHA statement, Mayo Clinic and the CDC.

Condition	Contagious?	How it usually differs	How doctors usually confirm
Kawasaki disease	No	Fever of five days or more; red eyes without discharge; peeling fingertips later; fever does not respond to antibiotics	Clinical criteria plus blood tests and heart ultrasound; no single test
Scarlet fever	Yes (strep bacteria)	Sandpaper-textured rash; sore throat prominent; responds to antibiotics	Throat swab or rapid strep test
Measles	Yes, highly	Cough, runny nose and eye redness before the rash; tiny white spots inside the mouth; rash starts on the face	Blood or swab test; vaccination history
Adenovirus infection	Yes	Sticky eye discharge; sore throat and cough; usually shorter fever	Swab test for the virus
Multisystem inflammatory syndrome in children (MIS-C)	No, but follows a contagious infection	Tends to affect older children; stomach pain, vomiting and diarrhea common; often low blood pressure	Evidence of recent COVID-19 infection plus inflammation markers

Two things stand out. First, the contagious illnesses in the list have confirmatory tests, which is exactly why doctors run them: ruling out scarlet fever or adenovirus narrows the field. Second, the illness that most resembles Kawasaki disease in its mechanism, MIS-C, is likewise not contagious in itself. The CDC describes MIS-C as a post-infectious inflammatory condition. The virus that precedes it spreads; the inflammatory syndrome that follows does not, which is the same logic that applies to Kawasaki disease.

The overlap is real enough that the distinction can take a day or two to settle, and some children are treated for the possibility of both while results come back.

Does Kawasaki disease go away? What the following weeks usually look like

Yes, the inflammation itself goes away. Even without treatment, the fever eventually burns out and the rash, red eyes and swelling resolve over a few weeks. The illness is what doctors call self-limited. The reason it is treated urgently is not to make it stop, but to protect the coronary arteries while it runs its course.

The numbers behind that urgency come from the AHA statement. Left untreated, about one in four children develop coronary artery aneurysms. With IVIG given within the first ten days of fever, that figure falls to roughly 4 percent. Those two numbers, more than anything else, explain why the whole system moves quickly once the diagnosis is suspected.

The follow-up rhythm is built around the timing of artery changes. The AHA recommends an echocardiogram at diagnosis, another at one to two weeks, and another at four to six weeks. Children whose scans stay normal through that final check are generally considered to have escaped the main complication, and how much further follow-up they need is a decision for the cardiologist. Children whose arteries did change enter a longer program of monitoring, with medicines and scan intervals set by the size of the changes and how they evolve. Aneurysms can shrink and remodel over months in many children, but the AHA notes that a vessel wall that has been badly stretched does not return to fully normal, which is why follow-up in this group continues into adulthood.

Two practical points often catch families out. Live vaccines such as measles and chickenpox are usually delayed for about eleven months after IVIG, because the donated antibodies can blunt the vaccine response, a recommendation found in both the AHA statement and CDC immunization guidance. And recurrence, while possible, is uncommon: the AHA cites a rate of about 3 percent in Japan, with lower figures reported elsewhere.

Is Kawasaki disease contagious to siblings? Myths people often get wrong

Some misunderstandings about this illness are harmless. Others lead to unnecessary isolation, guilt, or worse, a missed follow-up scan. These are the ones worth correcting.

Myth: siblings will catch it. They cannot. A brother or sister does carry a higher-than-average risk, but the AHA attributes that to shared genetic susceptibility, and the absolute risk remains low. Keeping the sibling home from school protects nobody.

Myth: it is caused by carpet cleaning or a dirty house. An early study noticed some families had recently shampooed carpets. Later research did not confirm any link, and no major health body lists household cleaning as a cause. It is not a hygiene problem.

Myth: it is an infection, so antibiotics will fix it. Antibiotics kill bacteria. There is no bacterium here to kill, which is one reason a fever that ignores antibiotics raises a doctor’s suspicion.

Myth: only Asian children get it. Rates are highest in children of East Asian ancestry, but the illness occurs in every population, and the majority of cases in the United States are in children without that ancestry.

Myth: aspirin is never safe for children, so the treatment must be a mistake. The general caution is real; the NHS and AHA both describe this illness as a recognized exception under specialist supervision, with the balance of benefit and risk weighed by the team.

Myth: once the fever is gone, the danger is over. Aneurysms typically form in the weeks after the fever settles. The four-to-six-week scan is the one that answers the question that matters most.

Myth: it is the same thing as MIS-C. They share features and some treatments, and both are noncontagious inflammatory responses, but the CDC describes MIS-C as a distinct condition following COVID-19 infection, with its own age pattern and typical symptoms.

Myth: a parent caused it. Nothing in the evidence supports this. The cause is unknown, and it is not a consequence of feeding, parenting, vaccination or care choices.

Questions to ask your care team about Kawasaki disease

Ward rounds move fast, and the most important questions tend to surface at three in the morning when no one is available to answer them. Writing them down helps. The list below is a starting point; the answers belong to the team caring for your child, who know the specific picture.

• Is the diagnosis complete or incomplete, and what would make you more or less confident over the next day or two?
• What did the first heart ultrasound show, and when are the next scans scheduled?
• How many days into the fever are we, and how does that timing affect the treatment plan?
• What should we expect during and after the IVIG infusion, and what reactions should we tell a nurse about immediately?
• If the fever comes back after the infusion, what are the options you would consider?
• Why is aspirin being used here when it is normally avoided in children, and what signs should we watch for at home?
• Are there other infections you are still testing for, and does a positive result change anything?
• Which vaccines need to be delayed, and for how long, and who will update the schedule?
• When can our child go back to daycare or school, and are there activities to avoid in the coming weeks?
• Who do we call if something worries us after discharge, and what number works at night and on weekends?
• If the arteries were affected, what does long-term follow-up look like, and who coordinates it?

It helps to ask one person to be the family’s note-taker during conversations, and to ask the team to write down the follow-up scan dates before discharge. Families consistently report that the hardest part of this illness is the waiting between scans, and a clear written plan makes that wait easier to carry.

When to call your doctor about Kawasaki disease

Before a diagnosis, the single most useful signal is time. A child with a fever that has lasted five days, particularly a young child, should be seen by a clinician even if nothing else seems dramatic, and sooner if the fever is accompanied by a rash, red eyes, cracked lips or swollen hands and feet. Call urgently for an infant under six months with a persistent unexplained fever, because the classic features are often absent at that age.

After diagnosis and discharge, contact the care team the same day if the fever returns, if your child develops new pain or swelling in the joints, if bruising or bleeding appears that seems out of proportion, or if there has been contact with chickenpox or influenza while aspirin is being taken, since the NHS advises that this combination needs a prompt conversation with the prescribing team. Also call if your child refuses fluids, produces very little urine, or becomes unusually drowsy or hard to rouse.

Call emergency services or go to the nearest emergency department without waiting if your child has any of the following:

• Chest pain, or a small child who is suddenly inconsolable while pale, gray, sweaty or clammy
• Breathing that is fast, labored or accompanied by grunting
• Fainting, collapse, or lips and face turning blue
• A seizure
• Severe abdominal pain with vomiting
• Signs of a serious allergic reaction during or after an infusion, such as facial swelling, hives spreading rapidly, or difficulty breathing

These signs are rare, and listing them is not meant to frighten. They are the situations in which minutes matter, and the only right response is the emergency system rather than a phone call or a search engine. For everything less urgent, the team who treated your child is the right first call, and no question about a change in your child is too small to ask.

Frequently asked questions

Is Kawasaki disease contagious to siblings or other children?

No. Kawasaki disease cannot be passed from one child to another, even within the same household. Siblings do carry a somewhat higher risk, but the AHA attributes that to shared genetic susceptibility rather than transmission, and the absolute risk stays low. A brother or sister can attend school and daycare as usual, and parents can care for the affected child without any special precautions.

Does Kawasaki disease go away on its own?

Yes, the inflammation is self-limited and resolves over a few weeks even without treatment. The reason doctors treat it urgently is to protect the coronary arteries while it runs its course, because untreated children face about a one-in-four chance of artery aneurysms according to the AHA. Follow-up heart scans over four to six weeks confirm whether the arteries came through unaffected.

When does the rash typically appear in Kawasaki disease?

The rash usually shows up within the first five days of fever, according to both the AHA scientific statement and Mayo Clinic. It varies widely in appearance, from blotchy red patches to a fine measles-like spread, and is often prominent in the diaper area. Peeling of the fingertips and toes is a separate, later change that typically begins in the second or third week.

Can adults get Kawasaki disease?

Rarely, yes, and case reports exist, but Kawasaki disease in adults is unusual and most adults with a similar picture turn out to have another condition, such as a drug reaction, a toxin-mediated infection or a post-infectious inflammatory syndrome. Adults who had the illness as children may need lifelong cardiology follow-up if their coronary arteries were affected, a decision guided by their original scan results.

How many kids get Kawasaki disease each year?

In the United States the AHA statement estimates roughly 20 cases per 100,000 children under five each year, while Japan records more than 250 per 100,000 in the same age group. Cases are rare in any single school or community, yet the illness remains the leading cause of acquired heart disease in children in developed countries because of its effect on the coronary arteries.

What causes Kawasaki disease?

The cause is unknown. The leading theory, supported by the age pattern, seasonal clustering and strong genetic influence, is that a common environmental trigger, probably an ordinary germ, sets off an exaggerated immune reaction in genetically susceptible children. No specific virus or bacterium has been confirmed, and there is no established link to diet, parenting, household cleaning products or routine vaccines.

Why is aspirin used in Kawasaki disease when children normally should not take it?

Aspirin is usually avoided in children under sixteen because of a rare complication affecting the liver and brain. The NHS and AHA both describe Kawasaki disease as a recognized exception, used under specialist supervision, first for its anti-inflammatory effect and later to make platelets less sticky while their numbers are high. All decisions about its use and duration rest with the treating team.

Can Kawasaki disease come back after treatment?

It can, but recurrence is uncommon. The AHA statement cites a recurrence rate of about 3 percent in Japan, with lower figures reported in other countries. A fever returning within the first days after IVIG is more often a sign that the first treatment did not fully control the inflammation, which is why the team asks families to report any new fever promptly.

Why are vaccines delayed after treatment for Kawasaki disease?

IVIG contains a large pool of donated antibodies that can neutralize live vaccines such as measles and chickenpox before the child’s own immune system responds to them. For that reason the AHA and CDC guidance recommend delaying these particular vaccines for about eleven months after the infusion. Inactivated vaccines are generally not affected, and the care team will advise on the child’s updated schedule.

Is Kawasaki disease the same as MIS-C?

No, though they resemble each other. Both are noncontagious inflammatory responses that can affect the heart and share some treatments. The CDC describes MIS-C as a distinct syndrome that follows COVID-19 infection, more often affects older children, and commonly brings stomach pain, vomiting and low blood pressure. Doctors sometimes need a day or two of test results to tell the two apart.

References

• CDC – About Kawasaki Disease
• NHS – Kawasaki disease
• MedlinePlus – Kawasaki disease

This article is for general information only and is not a substitute for professional medical advice. Please consult a qualified doctor about your individual situation.