When Does Numbness or Tingling in the Feet Call for a Peripheral Neuropathy Evaluation?

The sock feels bunched under the ball of her foot. She stops on the stairs, pulls it straight, keeps walking. The fold is still there. By the third time she checks, she realizes the sock is fine. The feeling is coming from the foot itself.

Moments like that are how most people first meet peripheral neuropathy: not with pain, but with something quietly wrong at the edges of the body. The question that follows is practical and a little anxious. Is this nothing, or is this the start of something? Knowing when to see a neurologist for numbness is less about panic and more about pattern: how fast it arrived, where it sits, and what else came with it.

This explainer walks through what the evaluation involves, who is usually seen quickly, who is asked to watch for a while, and which signs should never wait for a routine appointment.

What is peripheral neuropathy, and why do the feet feel it first?

Peripheral neuropathy is damage to the nerves outside the brain and spinal cord, the long cables that carry sensation in and movement commands out. The National Institute of Neurological Disorders and Stroke estimates that more than 20 million people in the United States live with some form of it, and the true number is probably higher because milder cases are never tested.

Why the feet? Distance. The nerve fibers that serve your toes run from the lower spine all the way down the leg, making them the longest cells in the body. A process that harms nerves throughout the body, such as high blood sugar, will show up first at the far end of the longest wire, which is why symptoms so often begin in both feet at once and only later climb toward the ankles and shins. Clinicians call this a stocking distribution. When hands join in later, it becomes a stocking-and-glove pattern.

Nerves also come in sizes. Large fibers carry vibration, position sense, and the signals that move muscles. Small fibers carry pain, temperature, and the autonomic functions you never think about, such as sweating and blood pressure adjustment. Small fibers tend to be injured earlier, which is one reason burning, pins and needles, and cold-water sensations often precede true loss of feeling or weakness.

The damage itself takes two main forms. Sometimes the insulating sheath around the nerve, called myelin, breaks down and signals slow. Sometimes the fiber inside, the axon, degenerates and signals fade. The distinction matters because it steers testing and points toward different causes, which is why an evaluation is worth more than a guess.

How do I know if my numbness is serious?

Three questions do most of the sorting, and clinicians run through them almost reflexively.

First, how fast did it arrive? Numbness that appears over seconds or minutes is a different animal from numbness that has crept in over months. Sudden onset, particularly on one side of the body, is treated as a possible stroke until proven otherwise. The CDC’s stroke warning signs list sudden numbness or weakness of the face, arm, or leg, especially on one side, alongside confusion, trouble speaking, vision loss, and loss of balance. Numbness that develops over hours to a few days and climbs the legs is also urgent, because it can signal an acute inflammatory nerve condition.

Second, where exactly is it? Symmetric numbness in both feet suggests a body-wide process affecting the longest nerves. Numbness confined to one nerve’s territory, such as the outer foot or the top of the foot, points more toward a pinched or compressed nerve. Numbness in the inner thighs, groin, and around the seat, sometimes called saddle numbness, is an emergency because it can indicate compression of the nerve roots at the base of the spine.

Third, what came with it? Numbness alone, stable for weeks, in someone with a known risk factor such as diabetes, is common and usually handled first in primary care. Numbness plus new weakness, foot drop, a change in walking, unexplained weight loss, or a wound on the foot you did not feel changes the calculation.

Duration rounds out the picture. MedlinePlus advises seeing a clinician when numbness has no obvious cause, lasts beyond a short episode, or keeps returning. Pins and needles from sitting cross-legged resolve in minutes. Anything that has settled in for weeks deserves a conversation.

When to see a neurologist for numbness: the thresholds most guidelines share

Most people do not go straight to a neurologist, and most do not need to. In the NHS pathway and in typical US practice, the first stop is a primary care clinician who examines the feet, checks reflexes and sensation, and orders blood tests that catch the most common and most fixable causes. A large share of evaluations begin and end there.

Referral to a neurologist, a physician who specializes in disorders of the nervous system, tends to follow when one or more of these thresholds is crossed:

• The cause is not clear after initial blood tests and examination.
• Symptoms are progressing over weeks to months rather than holding steady.
• Weakness, muscle wasting, or a change in gait accompanies the sensory change.
• The pattern is unusual: one-sided, patchy, affecting hands before feet, or involving the face.
• Autonomic symptoms appear, such as dizziness on standing, unusual sweating changes, or bowel and bladder disturbance.
• Nerve conduction testing or other specialized studies are needed to characterize the damage.
• Someone under roughly middle age has a neuropathy with no obvious explanation, since inherited and inflammatory causes become more likely.

Notice what is not on that list: severity of discomfort alone. Painful feet are miserable, but pain intensity does not track well with how dangerous the underlying process is. A person with mild, painless numbness that is climbing toward the knees may need a specialist sooner than someone with intensely burning but stable soles.

None of this is a self-service decision. The point of knowing the thresholds is to describe your symptoms clearly and ask whether referral makes sense, then let the clinician who examined you weigh the whole picture.

Numbness and tingling in feet causes: which neurological conditions are involved?

The list of causes is long, which is precisely why an evaluation matters. Mayo Clinic groups them into a handful of families, and each leaves a slightly different fingerprint on the nervous system.

Metabolic causes lead the field. Diabetes is by far the most common, followed by chronic kidney disease, thyroid disorders, and nutritional deficiencies, especially vitamin B12. Alcohol, when used heavily over years, damages nerves both directly and by depleting the B vitamins they depend on.

Autoimmune and inflammatory conditions form a second family. Guillain-Barré syndrome is a rapidly evolving attack on peripheral nerves that can progress over days; its slower cousin, chronic inflammatory demyelinating polyneuropathy, unfolds over months. Systemic autoimmune diseases such as lupus, rheumatoid arthritis, and Sjögren’s syndrome can involve nerves as one part of a wider picture.

Infections come next: shingles, HIV, hepatitis C, and Lyme disease among them. Then medicines, most notably some chemotherapy agents and certain antibiotics, which can injure nerves as a recognized side effect that the prescribing team monitors for.

Compression and injury account for the focal cases. A herniated disc pressing on a nerve root, or a nerve squeezed as it passes a narrow tunnel at the knee or ankle, produces numbness in that nerve’s territory rather than in both feet symmetrically.

Inherited neuropathies, such as the Charcot-Marie-Tooth group, often declare themselves with high arches, weak ankles, and a family story of tripping. Rarer still are neuropathies tied to abnormal proteins, certain cancers, or toxins such as heavy metals.

Even after a thorough work-up, NINDS notes that a meaningful proportion of cases remain idiopathic, meaning no cause is found. That is frustrating, but it is also information: it rules out the dangerous and the reversible, and it lets the focus shift to protecting the feet and managing symptoms.

Diabetes and the feet: why nerve damage is so common and so quiet

The National Institute of Diabetes and Digestive and Kidney Diseases estimates that about half of all people with diabetes have some form of nerve damage. Many do not know it, because the earliest change is not pain but a gradual dulling that the brain simply stops noticing.

The mechanism is cumulative. Sustained high blood glucose damages the tiny blood vessels that nourish nerve fibers and disrupts the chemistry inside the nerves themselves. Over years, the longest fibers fail first, which is why the classic diabetic neuropathy is symmetric, starts in the toes, and slowly ascends. High blood pressure, abnormal cholesterol, smoking, and long duration of diabetes each raise the odds.

The danger is indirect. A numb foot cannot report a blister from a new shoe, a pebble in a sock, or a burn from a heating pad. Small injuries go unnoticed, become ulcers, and can become infected. This is why the NHS advises that people with diabetes have their feet examined by a clinician at least once a year, and why a foot wound that you cannot feel is one of the strongest reasons to be seen promptly rather than at the next routine visit.

For someone with diabetes, then, the evaluation question shifts slightly. The likely cause is already in view; what the visit establishes is how far the damage has progressed, whether another cause is contributing, and what protective routine fits. A simple annual check of sensation with a fine nylon filament and a tuning fork, alongside a look at skin, pulses, and footwear, catches most problems early.

Blood sugar control slows progression, and that is the lever people hold most directly. How to tighten it, and with what, is a conversation for the prescribing team rather than a decision to make alone.

Vitamin B12, alcohol, and medicines: the causes that can often be corrected

Among the crowd of causes, a few stand out because catching them changes the outcome. Vitamin B12 deficiency heads that group.

B12 is needed to maintain the myelin sheath around nerves. When it runs low, numbness and tingling in the feet and hands appear, sometimes with balance trouble, fatigue, and memory changes. The NIH Office of Dietary Supplements notes that neurological damage from prolonged deficiency may become irreversible, which is exactly why clinicians test for it early rather than late. The same source lists people at higher risk: older adults whose stomachs absorb the vitamin less efficiently, people with pernicious anemia or after certain stomach or intestinal surgeries, those on strict vegan diets without supplementation, and people taking long-term acid-suppressing medicines or metformin, both of which can reduce absorption.

Two details matter in practice. First, B12 deficiency can damage nerves before any anemia shows on a routine blood count, so a normal count does not rule it out. Second, borderline results sometimes need a follow-up test of a related marker to settle the question. Whether and how to correct a deficiency is a prescribing decision that depends on the cause of the shortfall.

Alcohol is the second correctable contributor. Sustained heavy drinking is toxic to nerves and crowds out the thiamine and other B vitamins they need; reducing intake can stop progression, although recovery of feeling is slow and incomplete in many cases.

Medicines round out the group. Some chemotherapy drugs, certain antibiotics used long term, and a few other agents are known to affect nerves. If you suspect a medicine, the right move is to raise it with the prescriber, never to stop or adjust it yourself; the team weighs the nerve risk against what the medicine is treating.

Peripheral neuropathy tests: what actually happens at the evaluation

The evaluation is more listening than machinery. Expect the first fifteen minutes to be questions: when it started, whether it is climbing, whether both sides match, what medicines you take, how much you drink, what runs in your family, and whether you have noticed weakness, tripping, dizziness on standing, or wounds you did not feel.

The physical exam then maps the problem. A clinician touches the feet with a fine nylon filament to test pressure sensation, holds a vibrating tuning fork against the toe and ankle, checks whether you can tell which way a toe is being moved with your eyes closed, and taps the ankle and knee for reflexes, which often fade early. Strength testing looks for weakness in lifting the foot or spreading the toes. Watching you walk, including on heels and toes, tells its own story.

Blood tests come next and are chosen to catch the common causes: glucose and hemoglobin A1c for diabetes, vitamin B12, thyroid and kidney function, a blood count, and often a protein screen. Depending on the story, tests for inflammatory markers, infections, or autoimmune conditions follow.

If the picture is unclear or the pattern is unusual, nerve conduction studies and electromyography are the standard next step, as the NHS describes. Small electrodes deliver mild electrical pulses along the nerve and time how fast and how strongly the signal travels; a fine needle then records electrical activity inside muscles. The sensations are odd rather than painful, and the whole session typically runs under an hour.

Nerve conduction studies mainly assess large fibers. When small-fiber neuropathy is suspected and the standard studies are normal, a tiny skin biopsy from the leg can count nerve fiber density under a microscope, an approach Cleveland Clinic describes in its overview. Imaging of the spine is reserved for cases where a compressed nerve root or spinal cord problem is on the table.

Which patterns of numbness point where? A quick comparison

Patterns are not diagnoses, and the same distribution can arise from different causes. Still, the way numbness is laid out on the body is the single most useful clue a clinician has, and it sets the pace of the evaluation. The table below summarizes how these patterns are usually read in primary care and neurology, drawing on the descriptions in MedlinePlus, Mayo Clinic, and the NHS.

Pattern of numbness	What it often points toward	Usual pace of evaluation
Both feet, symmetric, slowly ascending over months	Length-dependent polyneuropathy, commonly diabetes, B12 deficiency, alcohol, or idiopathic	Routine appointment; blood tests first
One foot or one nerve territory, often after a position or injury	Compressed or entrapped nerve, or a nerve root from the spine	Routine to prompt, depending on weakness
Feet then hands, with weakness climbing over hours to days	Acute inflammatory neuropathy such as Guillain-Barré syndrome	Same-day emergency care
Sudden, one side of body, with face droop, speech or vision change	Stroke or transient ischemic attack	Call emergency services immediately
Inner thighs, groin, seat area, with bladder or bowel change	Compression of nerve roots at the base of the spine	Same-day emergency care
Patchy, asymmetric, different limbs at different times	Inflammatory, vasculitic, or infectious neuropathy	Prompt referral to neurology

Two cautions. Symmetry is reassuring about urgency but says nothing about reversibility; a slow symmetric neuropathy from B12 deficiency still needs timely correction. And a pattern can shift. Something that began in one foot and now involves both, or that has picked up speed, has moved rows and deserves a fresh look.

When to see a neurologist for numbness that has no pain: who is seen promptly and who waits

Painless numbness is often the case that lingers longest before anyone acts. It does not hurt, it does not wake you, and it is easy to file under aging or tight shoes. Yet the absence of pain tells a clinician nothing about how much nerve is involved. Large-fiber damage, which erodes vibration and position sense, is frequently painless and is exactly the kind that causes falls.

Who is usually seen promptly, whether by primary care or neurology, regardless of pain:

• Anyone whose numbness is spreading or accompanied by new weakness or a change in walking.
• People with diabetes who have a foot wound, a new deformity, or a foot that feels warm and swollen without pain.
• Those whose numbness came with dizziness on standing, fainting, or new bladder or bowel changes.
• Younger adults with a symmetric neuropathy and no obvious cause, where inherited or inflammatory conditions are more likely.
• People on medicines known to affect nerves, so the prescribing team can reassess.

Who is more often asked to watch and wait, with a plan:

• Someone with intermittent tingling that clearly follows a posture, resolves within minutes, and has no other features.
• A person with a known, stable cause already under management, whose symptoms have not changed.
• Someone whose initial blood tests have identified a correctable cause and who is being rechecked after that correction, since nerve recovery lags behind laboratory improvement.

Waiting is not the same as ignoring. It comes with a time frame, usually a few weeks to a few months, and clear instructions on what would bring the appointment forward. If you are asked to wait, ask what specifically should prompt you to call, and write it down. The clinician who examined you holds the decision, but you hold the observation post.

What the following weeks usually look like after a neuropathy work-up

The first visit rarely produces a full answer. What it produces is a plan, and knowing its shape reduces the anxiety of the gap.

Blood test results typically return within days. If they reveal a clear cause, such as untreated diabetes or a B12 deficiency, the next conversation is about correcting it, and follow-up is usually set to recheck both the lab value and the symptoms. Here expectations matter. Laboratory numbers can normalize within weeks, but nerves regrow slowly, roughly a millimeter a day at best, and MedlinePlus and Mayo Clinic both describe recovery of sensation as gradual and sometimes partial. Symptoms holding steady rather than improving is often the first sign that the cause has been addressed.

If the blood work is unrevealing or the pattern is unusual, a referral for nerve conduction studies and electromyography follows. Depending on local scheduling, that can take a few weeks. The results usually go back to the referring clinician or neurologist, who then either names the type of neuropathy, orders a targeted second round of tests, or, in a proportion of cases, concludes that the cause is idiopathic.

Running alongside all of this is foot protection, which starts on day one regardless of cause: daily visual inspection of the soles with a mirror if needed, well-fitting shoes, no barefoot walking, water temperature checked with the hand rather than the foot. For anyone with diabetes, the NHS annual foot review becomes a fixed appointment rather than an optional one.

Symptom management, if pins and needles or burning are disruptive, may begin during this period as well. The NHS notes that medicines used for nerve pain commonly take several weeks to show their effect, so an early follow-up is often scheduled to judge response rather than expecting immediate change.

How is peripheral neuropathy treated once the cause is known?

Treatment has two tracks that run at once: address the cause, and manage the symptoms. The first track matters more, because it is the only one that can halt progression.

Addressing the cause looks different for each family. In diabetes, it means tighter glucose control alongside attention to blood pressure and cholesterol. In deficiency states, it means replacing the missing vitamin under supervision. In alcohol-related neuropathy, it means reducing or stopping intake, ideally with support. In inflammatory neuropathies, treatments that calm the immune system are used and monitored by the specialist team. In medication-related cases, the prescriber weighs adjusting the offending medicine against what it is treating. None of these decisions belong to a magazine article; all of them belong to the clinician who knows your history.

Symptom management targets the pain and tingling that small-fiber damage produces. Ordinary painkillers help little, because the problem is faulty signaling rather than tissue injury. The NHS describes the classes typically used: certain antidepressants that dampen pain signaling in the spinal cord, and anti-seizure medicines that quiet overexcitable nerves. Both work on how the signal is processed rather than on the damaged nerve itself, and both usually take weeks to show their full effect. Topical treatments applied to the skin are an option for localized discomfort. Which class fits depends on your other conditions and medicines, and any change is the prescriber’s call.

Non-drug measures round out the picture: physical therapy for balance and strength, properly fitted footwear or orthotics, and for some people a transcutaneous electrical nerve stimulation device. Evidence for many supplements marketed for neuropathy is limited or inconsistent, and the NIH Office of Dietary Supplements is the honest place to check what studies actually show before spending on them.

What people often get wrong about numb feet

Myth: if it does not hurt, it is not doing harm. Painless loss of sensation is what leads to unnoticed foot injuries and to falls, because the brain has lost its map of where the feet are. Painless is not harmless.

Myth: numb feet are just part of getting older. Age does modestly reduce vibration sense at the toes, but symmetric numbness that climbs toward the ankles is not normal aging. Assigning it to age is the single most common reason a correctable cause, such as B12 deficiency, goes unfound for years.

Myth: it must be poor circulation. Blood flow problems in the legs usually announce themselves with cramping pain on walking that eases with rest, cold or pale skin, and slow-healing wounds. Numbness and tingling are far more often nerve than vessel, though both can coexist, especially in diabetes, which is why the foot exam checks pulses too.

Myth: a normal blood count rules out B12 deficiency. Nerve damage can appear before anemia does. The vitamin level itself, and sometimes a related marker, is what the evaluation checks.

Myth: nerve conduction studies are the definitive test. They are excellent for large fibers and for locating compression, but they can be normal in small-fiber neuropathy. A normal study does not mean the symptoms are imagined; it narrows the search.

Myth: once the cause is fixed, feeling comes back quickly. Nerves regenerate slowly and sometimes incompletely. The realistic early win is that things stop getting worse.

Myth: a neurologist is the only clinician who can help. Most evaluations begin and end in primary care, and diabetes teams, podiatrists, and physical therapists each hold part of the picture. Referral is one tool among several, chosen when the pattern calls for it.

Questions to ask your care team

A good visit is shaped as much by what you ask as by what you are told. These questions tend to move the conversation forward without requiring you to already know the answers.

• Based on my pattern of numbness, does this look like a body-wide nerve process or a single compressed nerve?
• Which blood tests are you ordering, and which common causes do they cover?
• Is there anything in my current medicines that could be contributing, and who should review that?
• Do I need nerve conduction studies now, or would you wait for the blood results first?
• If no cause is found, what does that mean for me, and what would you want to recheck later?
• What specific changes should make me call before my next scheduled visit?
• How should I inspect and protect my feet day to day, and how often should a clinician examine them?
• If we treat the symptoms, what timeline should I expect before judging whether something is working?
• Would physical therapy for balance help me, given what you found on the exam?
• Is there anything about my walking or reflexes today that you would want to compare against at the next visit?

Bring a written note of when the symptoms started, how they have changed, and anything that seems to make them better or worse. If you have diabetes, bring your recent glucose records and your shoes; a clinician can learn a surprising amount from wear patterns. Ask for a copy of any nerve study report so it travels with you if you see someone else later.

Finally, if something you read has worried you, say so directly. Naming the fear lets the clinician address it, and the answer is very often more reassuring than the search results were.

When to call your doctor: the red flags for numbness

Most numbness in the feet is not an emergency. A specific short list is, and it is worth knowing cold.

Call emergency services immediately if numbness comes on suddenly, especially on one side of the body, or arrives with any of the CDC stroke warning signs: drooping on one side of the face, weakness of an arm or leg, slurred or confused speech, sudden loss of vision, or sudden severe loss of balance. Minutes matter for stroke treatment, and the numbness itself may be the only sign you notice first.

Seek same-day emergency care if numbness or weakness is climbing the legs over hours to a few days, particularly if it reaches the hands or affects breathing or swallowing; if you develop numbness in the groin, inner thighs, or seat area together with new difficulty controlling your bladder or bowels; or if numbness follows a significant injury to the back or neck.

Contact your clinician promptly, within days, if numbness has lasted more than a few weeks without an obvious cause; if it is spreading or affecting both feet symmetrically; if you notice new weakness, foot drop, tripping, or a change in how you walk; if you have diabetes and find a blister, cut, ulcer, or area of warmth and swelling on a foot you cannot feel; if you feel dizzy or faint on standing; or if you have started a medicine known to affect nerves. Do not stop any prescribed medicine on your own while waiting.

Everything else, the intermittent tingling that follows a posture and clears within minutes, can wait for a routine appointment, mentioned alongside whatever else you are there for. The clinician who examines you weighs the pattern, the pace, and the company the numbness keeps, and that judgment, not a list, decides what happens next.

Frequently asked questions

How do I know if my numbness is serious?

Speed, location, and company decide it. Numbness that arrives suddenly, sits on one side of the body, or comes with weakness, speech change, or bladder problems is serious and needs emergency care. Numbness in both feet that has crept in over weeks or months is rarely an emergency but still deserves an evaluation, because common causes such as diabetes and vitamin B12 deficiency are easier to manage when found early.

Should I see a neurologist for tingling in my feet?

Usually not as a first step. Most tingling is evaluated in primary care with an examination and blood tests that catch the common causes. A neurologist is typically brought in when the cause remains unclear, symptoms are progressing, weakness or gait changes appear, the pattern is unusual, or nerve conduction studies are needed. Your primary clinician decides whether and when that referral fits your situation.

What are the red flags for numbness?

Sudden onset, especially one-sided, with face droop, arm weakness, or speech trouble points to stroke and needs emergency services. Numbness or weakness climbing the legs over hours to days, numbness in the groin and seat area with bladder or bowel changes, numbness after a spinal injury, and a foot wound you cannot feel in the setting of diabetes all call for same-day care.

What neurological conditions cause numbness in the feet?

Diabetic neuropathy is the most common. Others include vitamin B12 deficiency, alcohol-related neuropathy, inflammatory conditions such as Guillain-Barré syndrome and chronic inflammatory demyelinating polyneuropathy, inherited neuropathies like the Charcot-Marie-Tooth group, nerve compression from a herniated disc or narrow tunnel, infections including shingles and HIV, and side effects of certain medicines. A proportion of cases remain unexplained even after full testing.

What are the symptoms neurologists say you should never ignore?

Sudden weakness or numbness on one side, sudden trouble speaking or understanding, sudden vision loss, sudden severe imbalance or a headache unlike any before, and new loss of bladder or bowel control. Each can signal a time-sensitive condition affecting the brain or spinal cord. Alongside these, rapidly progressing weakness in the legs over hours to days should be treated as urgent rather than watched.

What peripheral neuropathy tests will I have at the evaluation?

Expect a detailed history and a physical exam testing sensation with a fine filament and tuning fork, reflexes, strength, and walking. Blood tests typically cover glucose, vitamin B12, thyroid and kidney function, and a blood count. If the picture is unclear, nerve conduction studies and electromyography follow, and in suspected small-fiber neuropathy a small skin biopsy may be used. Spinal imaging is reserved for suspected nerve root compression.

Can numbness in the feet with no pain still be neuropathy?

Yes, and it is common. Large nerve fibers that carry vibration and position sense are often damaged without producing pain, and their loss is a major cause of unsteadiness and falls. Painless numbness in someone with diabetes is also how unnoticed foot injuries begin. Absence of pain says nothing about how much nerve is involved, so painless numbness that persists still warrants an evaluation.

Can vitamin B12 deficiency cause tingling in the feet?

It can, and it is one of the causes clinicians test for first. B12 maintains the myelin sheath around nerves, and deficiency produces numbness, tingling, and sometimes balance problems, occasionally before any anemia appears. Older adults, people on strict vegan diets, those with absorption problems, and people taking long-term acid-suppressing medicines or metformin are at higher risk. Correction is a prescribing decision based on the underlying cause.

How long does numbness last after the cause is treated?

Longer than most people expect. Nerves regenerate slowly, and both MedlinePlus and Mayo Clinic describe recovery as gradual and sometimes partial. Laboratory values may normalize within weeks while sensation lags months behind, and in long-standing damage some numbness may remain. The realistic early sign that a cause has been addressed is that symptoms stop progressing rather than vanish.

Is numbness in the feet a sign of poor circulation or nerve damage?

Far more often nerve damage. Circulation problems in the legs typically cause cramping pain on walking that eases with rest, cool or pale skin, and slow-healing sores, rather than tingling. The two can coexist, particularly in diabetes, which is why a foot examination checks both sensation and pulses. Your clinician can usually distinguish them with the exam and, if needed, simple tests.

References

• Peripheral Neuropathy – National Institute of Neurological Disorders and Stroke (NIH)
• Peripheral neuropathy – NHS
• Numbness and tingling – MedlinePlus Medical Encyclopedia
• Stroke Signs and Symptoms – CDC

This article is for general information only and is not a substitute for professional medical advice. Please consult a qualified doctor about your individual situation.