The Types of Arthritis Explained (and Osteoarthritis vs Rheumatoid)

Two neighbors, both in their sixties, both complaining about their hands. One can barely turn a key first thing in the morning, then loosens up by breakfast and forgets about it until she has been gardening for an hour. The other wakes with knuckles that feel wrapped in wet cement for most of the morning, and she is tired in a way that no amount of coffee fixes. Same word on both their charts: arthritis. Almost nothing else in common.

That single word covers well over a hundred conditions, according to the CDC, and lumping them together is one of the reasons so many people wait years for the right diagnosis. Cartilage wearing thin is not the same problem as an immune system attacking the joint lining, and neither is the same as uric acid crystallizing in a toe overnight.

This guide sorts the seven forms you are most likely to meet, shows how doctors tell them apart, and takes an honest look at the questions people actually type into a search bar at 2 a.m.

Why "the 7 types of arthritis" is a useful shortcut, not a medical category

Search for a definitive list and you will find sevens, fives, tens, and the occasional twelve. None of them is wrong, exactly. Medicine does not recognize an official roster of seven; it recognizes more than 100 conditions that involve joint pain, swelling, or stiffness, which the CDC groups under one umbrella term. The number seven simply reflects the handful that fill most clinic appointments.

Arthritis is also astonishingly common. The CDC estimates that about 53 million US adults, roughly one in five, have some form of doctor-diagnosed arthritis. The NHS puts the UK figure at around 10 million people. Behind those totals, though, sit very different diseases with very different rules.

The list used in this article covers the conditions that matter most in practice:

• Osteoarthritis, by far the most common
• Rheumatoid arthritis, the classic autoimmune form
• Psoriatic arthritis, linked to the skin condition psoriasis
• Gout, driven by uric acid crystals
• Ankylosing spondylitis, which targets the spine
• Juvenile idiopathic arthritis, which begins in childhood
• Septic arthritis, an infection inside the joint and a genuine emergency

Others deserve a mention. Lupus and reactive arthritis both inflame joints; fibromyalgia often appears on “types of arthritis” lists even though it does not damage joints at all. Knowing which family a condition belongs to tells you more than memorizing seven names ever will, so that is where we start.

Wear or war: the two families every type of arthritis belongs to

Picture a door hinge. One way it fails is slow and mechanical: decades of opening and closing grind away the smooth surfaces until metal scrapes metal. That is degenerative arthritis, and osteoarthritis is its only major member.

The other way it fails has nothing to do with use. Something attacks the hinge from inside, swelling it, heating it, corroding the surfaces even while the door sits still. That is inflammatory arthritis, and it includes rheumatoid, psoriatic, ankylosing spondylitis, juvenile idiopathic arthritis, and lupus. Gout is inflammatory too, though the trigger is a chemical crystal rather than a misdirected immune system. Septic arthritis sits in its own corner, caused by bacteria or other microbes.

The distinction matters because it changes almost everything downstream. Degenerative arthritis tends to hurt more after activity and ease with rest. Inflammatory arthritis does the opposite: stiffness is worst after sleep or sitting still and loosens with movement. The Mayo Clinic notes that rheumatoid stiffness is typically worse in the mornings and after inactivity, while osteoarthritis stiffness shows up on waking or after rest and usually lifts quickly.

Inflammatory types also spill beyond the joints. Fatigue, low-grade fever, weight loss, and eye or skin involvement point away from simple wear and toward an immune process. Osteoarthritis stays local; it does not make you feel systemically unwell.

When a clinician takes your history, this is the fork in the road they are listening for before they order a single blood test.

Osteoarthritis: what actually happens inside a worn joint

Cartilage is remarkable stuff, smoother than ice on ice, and it lets bones glide over each other for decades. In osteoarthritis that cushion gradually thins and roughens. The Mayo Clinic describes it as a whole-joint disease, not just cartilage loss: the underlying bone changes shape, small bony spurs form at the edges, the lining becomes mildly inflamed, and the ligaments and muscles around the joint weaken.

The NHS reports that osteoarthritis affects nearly 9 million people in the UK, making it the most common type by a wide margin. It favors the joints that carry load or work hardest: knees, hips, the base of the thumb, the finger joints nearest the nails, the big toe, and the lower back and neck.

Typical features include:

• Pain that builds during or after activity and settles with rest
• Brief stiffness after waking or sitting, usually easing within half an hour
• A grating or crackling sensation when the joint moves
• Hard, bony enlargement of finger joints rather than soft, warm swelling
• Loss of flexibility that creeps up over years

Age is the strongest risk factor, but osteoarthritis is not simply “getting old.” Previous injuries, occupations with repetitive kneeling or lifting, excess body weight (which multiplies the force through knees and hips), and inherited joint shape all contribute. The Mayo Clinic also lists female sex and certain metabolic conditions as risk factors.

Symptoms often come and go in flares tied to activity, weather, or nothing obvious at all. The X-ray can look dramatic while the person feels fine, or look mild while the person struggles on stairs. Imaging and pain do not always agree, which is why treatment is guided by how you function, not by a picture.

Rheumatoid arthritis: when the immune system attacks the joint lining

Every joint is wrapped in a thin membrane called the synovium, which produces the fluid that keeps it lubricated. In rheumatoid arthritis the immune system targets that membrane. It thickens, floods with inflammatory cells, and over time erodes cartilage and bone from the inside. The disease is systemic, meaning the same inflammation circulates through the whole body.

The NHS estimates that rheumatoid arthritis affects more than 400,000 people in the UK, that it most often begins between ages 40 and 50, and that women are about three times more likely to develop it than men. It usually starts in the small joints of the hands and feet and tends to be symmetrical: if the left wrist is swollen, the right one often is too.

Hallmarks that separate it from wear-and-tear pain:

• Morning stiffness lasting well beyond 30 minutes, sometimes hours
• Joints that feel warm, soft, and boggy rather than hard
• Fatigue, low mood, and a general sense of being unwell
• Symptoms that improve with movement instead of worsening

The Mayo Clinic notes that about 40% of people with rheumatoid arthritis experience signs and symptoms outside the joints, including the skin, eyes, lungs, heart, blood vessels, and nerves. Firm lumps called rheumatoid nodules can form under the skin near pressure points.

Timing changes outcomes. Erosions that show on imaging are permanent, and the disease is most active in its first couple of years. That is why guidelines in the UK, US, and Europe push for early referral to a rheumatologist when inflammatory arthritis is suspected, ideally within weeks of symptoms starting rather than months.

Osteoarthritis vs rheumatoid arthritis: the side-by-side comparison

Put the two most common diagnoses next to each other and the differences stop being subtle. This is the comparison clinicians run through in their heads during the first five minutes of an appointment.

Feature	Osteoarthritis	Rheumatoid arthritis
Underlying process	Cartilage breakdown and bone remodeling	Autoimmune inflammation of the joint lining
Typical onset	Gradual, over years, usually after 50	Weeks to months, commonly ages 40 to 50
Joints involved	Knees, hips, thumb base, fingertip joints, spine	Knuckles, wrists, toes, often symmetrical
Morning stiffness	Brief, typically under 30 minutes	Prolonged, often over an hour
Effect of activity	Pain worsens with use	Stiffness eases with movement
Swelling	Hard, bony enlargement	Soft, warm, tender
Whole-body symptoms	None	Fatigue, fever, weight loss, organ involvement
Blood tests	Usually normal	Inflammatory markers and antibodies often raised

Blood tests deserve a caveat. Some people with rheumatoid arthritis test negative for the classic antibodies, and mildly raised inflammatory markers can appear in many unrelated situations. No single test settles it; the diagnosis rests on pattern, examination, imaging, and labs together.

One more wrinkle: the two can coexist. A person with long-standing rheumatoid arthritis may develop secondary osteoarthritis in joints the inflammation damaged years earlier, so a knee that hurts today can carry both stories.

Psoriatic arthritis: the type that starts with the skin

Most people know psoriasis as scaly, silvery patches on the elbows, knees, or scalp. Fewer know that the same immune process can move into the joints. The Cleveland Clinic reports that psoriatic arthritis affects about 30% of people who have psoriasis, and that joint symptoms usually appear years after the skin disease, though in a minority the joints come first.

What makes it distinctive is how untidy it is. Rheumatoid arthritis likes symmetry; psoriatic arthritis often picks a single knee, two fingers on one hand, and a toe on the other foot. Inflammation also targets places rheumatoid rarely touches:

• Entire fingers or toes swelling like sausages (called dactylitis)
• Tendon and ligament attachments, especially the heel and the sole of the foot
• The spine and pelvis, causing inflammatory back pain
• Nails, which pit, thicken, or lift from the nail bed

Fatigue and eye inflammation are common companions. The Cleveland Clinic notes that psoriatic arthritis is also associated with a higher risk of cardiovascular disease, obesity, and mood disorders, which is one reason rheumatologists look at the whole person rather than the joint alone.

Diagnosis can be slow because blood tests are frequently normal and the skin patches may be tiny and hidden in the scalp, behind the ears, or in the navel. If you have any history of psoriasis, even a small patch decades ago, mention it whenever a joint or a heel starts hurting. That single sentence can shorten the path to a diagnosis considerably.

Gout: why uric acid crystals make a big toe feel like it is on fire

Gout has a reputation as a punchline, the disease of Georgian kings and too much port. The reality is a chemistry problem. Uric acid is a normal waste product from breaking down compounds called purines. When blood levels run high for long enough, needle-shaped crystals form inside a joint, and the immune system reacts to them the way it would to shards of glass.

The attack is unmistakable. The NHS describes sudden, severe pain, commonly in the big toe but also the ankle, knee, fingers, or wrist, with the joint turning hot, red, swollen, and so tender that a bedsheet feels unbearable. Attacks often begin at night and, according to the NHS, typically last five to seven days before settling on their own.

Several factors nudge uric acid upward:

• Kidneys that clear uric acid slowly, which is largely inherited
• Diets heavy in red meat, organ meats, shellfish, and beer
• Sugary drinks sweetened with fructose
• Excess body weight and certain blood pressure conditions
• Some medicines, which a prescriber can review

Between attacks the joint feels completely normal, which tempts people to ignore the condition. That is a mistake. Repeated attacks damage the joint, and untreated high uric acid can form chalky deposits called tophi under the skin and around joints. The NHS also links gout with kidney stones.

Gout is one of the most controllable forms of arthritis. Long-term medicines that lower uric acid, combined with dietary changes, can reduce attacks dramatically, though the choice and timing belong with the prescribing clinician. A confirmed diagnosis, ideally by finding crystals in joint fluid, is worth insisting on before committing to years of treatment.

Ankylosing spondylitis: inflammatory back pain that eases when you move

Back pain that improves with rest is the ordinary kind. Back pain that is worst at 4 a.m., drives you out of bed, and gets better once you are walking around is a different animal, and it is the signature of ankylosing spondylitis, now often grouped under the broader label axial spondyloarthritis.

The condition inflames the joints of the spine and the sacroiliac joints where the spine meets the pelvis. Over years, uncontrolled inflammation can stimulate new bone growth that fuses vertebrae together, stiffening the spine. The NHS notes that symptoms usually begin in the late teens or twenties, which is precisely why it gets missed: young, otherwise healthy people are told they have pulled a muscle.

Clues that point toward an inflammatory cause include:

• Back or buttock pain lasting more than three months
• Onset before age 45
• Morning stiffness that lasts longer than 30 minutes
• Waking in the second half of the night with pain
• Improvement with exercise, not with rest

The disease does not stay in the spine. The NHS lists inflammation of the eye (uveitis), which causes a painful red eye and blurred vision, as a common complication; hips, shoulders, heels, and chest wall can be involved; and there is an association with inflammatory bowel disease and psoriasis. A specific genetic marker is found in most people with the condition but also in many healthy people, so it supports a diagnosis without confirming one.

Movement is medicine here in a literal sense. Regular stretching, posture work, and aerobic activity are core treatment, not an optional extra, alongside medicines that dampen inflammation.

Juvenile idiopathic arthritis: yes, children get arthritis too

A limp that comes and goes, a knee that looks puffy after a nap, a toddler who suddenly refuses to climb stairs. Parents rarely think of arthritis, and neither, at first, do many clinicians. Yet juvenile idiopathic arthritis is the most common chronic rheumatic disease of childhood, defined as joint inflammation beginning before age 16 and lasting at least six weeks with no other explanation. “Idiopathic” is medical shorthand for “we do not know the cause.”

It is not one disease but a group. Some children have a single swollen joint, often a knee, and may never develop more. Others have several joints involved in a pattern resembling adult rheumatoid arthritis. A smaller group has a systemic form with daily spiking fevers and a salmon-colored rash that appears and vanishes with the fever.

Two features make pediatric arthritis particularly worth catching early:

• Growing bones respond to inflammation by growing unevenly, so one leg can end up longer than the other
• Silent eye inflammation is common in certain subtypes and can damage vision without any redness or pain, which is why regular eye screening is standard care

Children are also poor witnesses to their own pain. They adapt, compensate, and stop doing the things that hurt rather than complaining. Watch for behavior instead of words: reluctance to walk in the morning, a preference for being carried, or a joint that looks different from its partner.

Most children do well with modern care, and a substantial number reach adulthood with the disease quiet. The goal, as with adult inflammatory arthritis, is to control inflammation before it changes a joint permanently.

Septic arthritis: the one type that is a same-day emergency

Every other condition in this article unfolds over weeks, months, or years. Septic arthritis unfolds over hours. Bacteria, usually arriving through the bloodstream from an infection elsewhere or through a wound or procedure, take hold inside the joint. Enzymes released by the infection and the immune response can destroy cartilage within days.

The picture is dramatic and usually involves a single joint, most often the knee or hip:

• Severe pain that makes any movement almost impossible
• Rapid swelling, heat, and redness
• Fever, chills, and feeling acutely unwell
• Inability to bear weight

People at higher risk include those with existing joint disease such as rheumatoid arthritis, artificial joints, diabetes, weakened immune systems, recent joint injections or surgery, and intravenous drug use. In the elderly and in people taking immune-suppressing medicines, fever may be muted, which makes the diagnosis harder and more dangerous.

Confirmation comes from drawing fluid out of the joint with a needle and examining it for bacteria and white cells. Treatment means intravenous antibiotics started promptly, often combined with draining the joint, and it takes place in a hospital setting.

The confusion that costs time is with gout, which can look almost identical: one hot, red, exquisitely painful joint. The two can even occur together. Nobody should try to tell them apart at home. A single joint that becomes severely painful, hot, and swollen within a day or two, especially with fever, needs urgent medical assessment the same day, not an appointment next week.

What is the most serious type of arthritis, and what organs can it affect?

Two questions, two different honest answers. In terms of immediate danger, septic arthritis wins without argument. It can permanently destroy a joint in days and, because the infection often originates in the bloodstream, it can be life-threatening.

In terms of long-term, whole-body impact, the systemic autoimmune forms carry the heaviest load: rheumatoid arthritis, lupus, and to a lesser extent psoriatic arthritis and ankylosing spondylitis. The Mayo Clinic documents that rheumatoid inflammation can involve the skin, eyes, lungs, heart, kidneys, salivary glands, nerve tissue, bone marrow, and blood vessels, and that people with the disease face higher risks of cardiovascular disease, lung scarring, osteoporosis, and lymphoma.

Organ involvement, by type, in broad strokes:

• Rheumatoid arthritis: eyes (dryness, inflammation), lungs (scarring, fluid), heart and blood vessels, low red cell counts
• Psoriatic arthritis: eyes, cardiovascular system, and metabolic health
• Ankylosing spondylitis: eyes, bowel, heart valves and rhythm in rare cases, lungs through restricted chest movement
• Gout: kidneys, through stones and reduced function, plus links to heart disease
• Lupus: kidneys, skin, blood, brain, heart lining

Osteoarthritis, by contrast, does not damage organs directly. Its seriousness is measured differently: in lost mobility, falls, sleep disruption, and the cascade that follows when a person stops moving. Calling it “the mild one” underestimates how much a painful knee can reshape a life.

The reassuring counterpoint is that the risks above describe what uncontrolled inflammation does over time. Modern management aims to keep disease activity low enough that most of these complications never develop, which is why early, sustained treatment is the single most important idea in inflammatory arthritis care.

Are there really 5 vegetables to avoid for arthritis?

Type that phrase into a search engine and you will be told to fear tomatoes, potatoes, peppers, eggplant, and sometimes spinach or corn. The theory is that nightshade vegetables contain compounds that inflame joints. The evidence for it, in mainstream medical literature, is essentially nonexistent. No controlled study has shown that removing nightshades improves any form of arthritis, and dietary guidance from the NHS, the CDC, and academic health systems does not advise avoiding them.

What the evidence does support is less dramatic but more useful:

• For gout, the NHS advises limiting purine-rich foods such as red meat, offal, oily fish, and shellfish, and cutting back on alcohol, especially beer, and sugary drinks. Vegetables, even purine-containing ones like spinach and asparagus, have not been shown to trigger attacks.
• For osteoarthritis, the strongest dietary lever is body weight, because every extra pound increases the force through knees and hips. Any pattern that helps you reach a healthy weight helps the joint.
• For inflammatory arthritis, a Mediterranean-style pattern rich in vegetables, olive oil, fish, whole grains, and legumes is associated with lower inflammatory markers, though the effect on disease activity is modest and it does not replace medical treatment.

Some people genuinely notice that a particular food worsens their symptoms. That experience is real, and a short, structured elimination with a dietitian is reasonable. Cutting out an entire vegetable group on the strength of a viral list, however, removes fiber, potassium, and antioxidants for no demonstrated benefit.

If a diet promise sounds like a cure, treat it the way you would any other cure claim for a chronic disease: with polite skepticism.

What is the best medicine for arthritis? Why the answer depends on the type

There is no best medicine for arthritis, for the same reason there is no best tool for a house: it depends entirely on what is broken. The approach splits along the same wear-versus-war line as everything else.

For osteoarthritis, the foundation is not a pill. Guidelines from the UK and US consistently rank exercise, strength training, and weight management first, because stronger muscles around a joint reduce the load through it and improve pain within weeks. Pain relievers and anti-inflammatory medicines, taken by mouth or rubbed on the skin, manage symptoms but do not change the cartilage. Injections of corticosteroid can calm a flare for weeks to a few months. When a joint is worn beyond what these can manage, joint replacement is one of the most successful operations in medicine.

For inflammatory arthritis, the logic reverses. Symptom relievers help you through the day, but the core treatment is medicine that quiets the immune process itself. Conventional disease-modifying drugs typically take several weeks to a few months to reach full effect, according to the NHS, which is why clinicians sometimes bridge with short courses of steroids. If those are insufficient, targeted biologic or small-molecule therapies block specific inflammatory signals; they tend to act faster but require screening for infections and regular monitoring.

Gout has its own two-part strategy: settling the acute attack, then lowering uric acid long term so crystals dissolve and attacks stop.

Which medicine, at what stage, and for how long is a decision for you and the prescribing clinician, weighing your other conditions, your kidneys and liver, your plans for pregnancy, and your priorities. What matters more than any single drug is getting the diagnosis right first, because the best medicine for the wrong arthritis is no medicine at all.

When to see a doctor about joint pain, and which signs should not wait

Most aching joints are not an emergency, and a few days of rest and gentle movement sort them out. Some patterns, though, deserve a scheduled appointment, and a small number need same-day care.

Book an appointment if you notice:

• Joint pain, swelling, or stiffness lasting more than a few weeks
• Morning stiffness that regularly lasts longer than 30 minutes
• Swelling in several joints, especially both hands or both feet
• Joint symptoms alongside fatigue, unexplained weight loss, rashes, or eye problems
• Back pain that wakes you in the early hours and eases with movement, particularly if you are under 45
• Repeated episodes of a sudden, hot, painful toe or ankle
• A child who limps, avoids using a limb, or has a joint that looks swollen

Seek urgent care the same day, through an emergency department or urgent line, for red-flag signs:

• A single joint that becomes severely painful, hot, red, and swollen over hours to a day or two
• Joint pain with fever, chills, or feeling very unwell
• Inability to move a joint or bear weight on it
• Joint pain after a deep wound, a recent joint injection, or surgery on that joint
• A painful red eye with blurred vision in anyone with known inflammatory arthritis

Bring notes. A week of jotting down when the stiffness starts, how long it lasts, and which joints are involved is worth more to a clinician than any single description you could give in the room. Photographs of a swollen joint taken at its worst help too, since flares have a habit of settling the morning of the appointment.

Early assessment costs an hour. Late diagnosis of inflammatory arthritis can cost a joint. That imbalance is why rheumatologists would rather see ten people who turn out to have osteoarthritis than miss one whose immune system is quietly eroding bone.

Frequently asked questions

What are the 7 types of arthritis?

The seven most commonly listed are osteoarthritis, rheumatoid arthritis, psoriatic arthritis, gout, ankylosing spondylitis, juvenile idiopathic arthritis, and septic (infectious) arthritis. The number is a convenience rather than an official classification; the CDC recognizes more than 100 conditions under the arthritis umbrella. Lupus, reactive arthritis, and fibromyalgia often appear on similar lists, though fibromyalgia does not actually damage joints.

What is the difference between osteoarthritis and rheumatoid arthritis?

Osteoarthritis is mechanical breakdown of cartilage and bone in a joint; rheumatoid arthritis is an autoimmune disease in which the immune system inflames the joint lining. Osteoarthritis hurts more with use and produces brief morning stiffness, while rheumatoid causes prolonged morning stiffness, warm soft swelling, symmetrical hand and foot involvement, fatigue, and sometimes problems in organs such as the eyes, lungs, and heart.

What is the most serious type of arthritis?

Septic arthritis is the most immediately dangerous because bacterial infection can destroy a joint within days and spread through the bloodstream. Among long-term conditions, rheumatoid arthritis and lupus carry the greatest whole-body risk, including cardiovascular disease and lung involvement. Osteoarthritis does not affect organs but can severely limit mobility, so seriousness depends on whether you measure by speed, systemic damage, or disability.

What organs are affected by arthritis?

Inflammatory types can affect organs beyond the joints. Rheumatoid arthritis can involve the eyes, lungs, heart, blood vessels, skin, nerves, and bone marrow. Ankylosing spondylitis and psoriatic arthritis commonly inflame the eye and are linked with bowel and cardiovascular disease. Gout affects the kidneys through stones and reduced function. Osteoarthritis stays confined to the joints and does not damage internal organs.

What are 5 vegetables to avoid for arthritis?

There are none supported by evidence. The popular list of nightshades, such as tomatoes, potatoes, peppers, and eggplant, comes from anecdote, and no controlled research shows that avoiding them improves any form of arthritis. Dietary advice that does have evidence includes limiting red meat, organ meats, shellfish, beer, and sugary drinks for gout, and maintaining a healthy weight for osteoarthritis. Vegetables of all kinds remain beneficial.

What is the best medicine for arthritis?

It depends on the type, which is why an accurate diagnosis comes first. Osteoarthritis is managed mainly with exercise, weight management, and symptom-relieving pain medicines. Inflammatory types such as rheumatoid and psoriatic arthritis need disease-modifying medicines that calm the immune system, which typically take weeks to months to work fully. Gout requires settling attacks and then lowering uric acid long term. Your prescribing clinician tailors the choice.

How do doctors tell which type of arthritis you have?

Mostly through your story and an examination: which joints hurt, how long morning stiffness lasts, whether movement helps or hurts, and whether you feel unwell overall. Blood tests for inflammation and specific antibodies support a diagnosis of rheumatoid arthritis but can be normal in psoriatic arthritis and ankylosing spondylitis. X-rays, ultrasound, or MRI show damage or inflammation, and drawing fluid from a joint confirms gout or infection.

Can you have more than one type of arthritis at the same time?

Yes, and it is common. Long-standing rheumatoid arthritis often leads to secondary osteoarthritis in joints the inflammation damaged. Gout and osteoarthritis frequently occur together in the same big toe or knee. Gout can also coexist with septic arthritis in a single joint, which is one reason a sudden hot, swollen joint should always be assessed rather than assumed to be a familiar flare.

Does cracking your knuckles or cold weather cause arthritis?

Knuckle cracking has not been shown to cause arthritis; the sound comes from gas bubbles in joint fluid, not damage. Cold or damp weather does not cause arthritis either, though many people report that existing joint pain feels worse in cold conditions, possibly through changes in pressure, muscle tension, and activity levels. Genuine risk factors include age, prior injury, excess weight, family history, and autoimmune conditions.

When should I see a doctor about joint pain?

Book an appointment if joint pain, swelling, or stiffness lasts more than a few weeks, if morning stiffness regularly exceeds 30 minutes, if several joints are involved, or if you also have fatigue, rashes, eye problems, or weight loss. Seek same-day urgent care for a single joint that becomes hot, red, and severely painful within a day or two, especially with fever, or if you cannot bear weight.

References

• CDC – Arthritis: About and Basics
• NHS – Arthritis
• NHS – Gout
• Cleveland Clinic – Psoriatic Arthritis

This article is for general information only and is not a substitute for professional medical advice. Please consult a qualified doctor about your individual situation.