Do Glasses Make Myopia Worse? Nearsightedness Myths Eye Doctors Hear Every Week

The mother in the exam chair has already made up her mind before the optometrist speaks. Her nine-year-old squinted at the board all fall, but she is worried that once he gets glasses, “his eyes will get lazy and he’ll never get them off.” Her own father said the same thing forty years ago. So did a neighbor last week.

Eye doctors hear a version of this conversation almost every day, and it sits at the top of a long list of myopia myths that shape how families handle nearsightedness. Some myths are harmless. Others delay correction a child needs, or push parents toward unproven fixes while the window for slowing progression quietly closes.

What follows is an honest, evidence-graded tour of what is known about nearsightedness, what is genuinely uncertain, and what eye care professionals wish more people understood before they sit down in that chair.

Do glasses make myopia worse? The evidence behind the biggest myopia myth

Start with the myth that fills the most exam-room minutes. The worry usually runs like this: glasses do the focusing for the eye, the eye stops “working,” and the prescription climbs as a result. It sounds mechanical and plausible. It is also not how myopia progresses.

Nearsightedness worsens because the eyeball grows too long from front to back during childhood, so light focuses in front of the retina instead of on it. That growth is driven by biology and by the visual environment, not by whether a lens sits in front of the eye. A pair of single-vision glasses simply shifts the focus back onto the retina. It does not weaken any muscle, because the muscles that move the eye and adjust the lens have nothing to do with the length of the eyeball.

The best test of the myth is the opposite experiment: what happens when children are deliberately given weaker glasses than they need? Researchers have tried this, on the theory that a little blur might discourage the eye from lengthening. The results pointed the other way. Under-corrected children progressed at least as fast, and in several studies faster, than fully corrected children. The idea that blur “trains” the eye toward normal vision has not held up.

Mainstream sources, including the Mayo Clinic and the NHS, describe glasses as the standard, safe correction for nearsightedness at any age, with no evidence that wearing them accelerates the underlying condition. The prescription that rises year after year is following the eye’s growth curve, which was already set in motion. The glasses are keeping up with it, not causing it.

That said, ordinary single-vision lenses do not slow the growth either. That is where a newer category of interventions comes in, discussed further below.

What actually happens inside a nearsighted eye

Picture the eye as a camera about the size of a large grape. Light passes through the clear front window (the cornea), then through a flexible lens, and lands on the retina, the light-sensitive layer at the back. For a sharp image, the distance from the front of the eye to the retina must match the focusing power of the cornea and lens.

In myopia, that distance is too long, or occasionally the cornea is too curved. Either way, light from distant objects comes to a focus in front of the retina and has spread out again by the time it arrives, producing blur. Close objects are unaffected because their light rays enter the eye at a steeper angle and naturally focus farther back, which is why a nearsighted child can read a phone comfortably yet cannot read a street sign.

The measurement you see on a prescription is given in diopters, a unit of focusing power. A minus sign indicates nearsightedness. Every additional diopter corresponds to roughly a third of a millimeter of extra eye length, which explains why small changes in growth show up as noticeable jumps in prescription. High myopia, often defined as around six diopters or more according to Cleveland Clinic, means the eye has stretched considerably beyond its intended length.

Why does stretching matter beyond blur? The retina and the tissues behind it are stretched along with the eyeball wall. Think of a balloon inflated further than intended: the material thins. That thinning is the reason eye care professionals care about the total amount of myopia a person accumulates, not just whether it can be corrected with a lens.

Most childhood myopia progresses because the eye keeps growing after it should have stopped. Adult-onset shifts are less common and often related to prolonged near work, certain medicines, or early cataract changes, which is why a sudden prescription change in adulthood deserves a proper exam rather than a stronger pair of glasses.

What are the main theories behind myopia, and which ones hold up?

Ask why nearsightedness has surged in one generation and you will hear several explanations. The evidence supports some far more than others.

Heredity. Children with one nearsighted parent are more likely to become nearsighted, and children with two are more likely still. Dozens of genes influencing eye growth have been identified. Genetics is real, but genes alone cannot explain why prevalence has roughly doubled in a few decades in some populations, since gene pools do not change that fast.

Near work. The oldest theory holds that hours spent focusing up close, whether on books or screens, nudge the eye to lengthen. Studies do find associations between heavy near work and myopia, but the effect is weaker and less consistent than many assume once time spent outdoors is accounted for.

Outdoor light. This is the theory with the strongest recent support. Children who spend more time outdoors are less likely to become nearsighted, and school-based programs that increased outdoor time reduced new cases. The leading explanation involves bright daylight stimulating dopamine release in the retina, a chemical signal that appears to restrain eye growth. Distance viewing outdoors may also play a role.

Peripheral defocus. A more technical theory proposes that the eye’s growth is guided by how light focuses at the edges of the retina, not just the center. This idea underpins the design of specialty lenses intended to slow progression.

The honest summary: myopia is a gene-environment condition. A child inherits a tendency, and modern childhood, with more schooling, more indoor hours, and less daylight, supplies the trigger. Blaming any single factor is one of the myopia myths that oversimplifies a genuinely complex process.

Myopia myths about screens, reading in dim light and eye strain

Screens take most of the blame in living rooms, so it is worth separating what tablets actually do from what people fear they do.

There is no good evidence that the light from a screen damages the eye or directly causes myopia. Screens are a form of near work, and near work has a modest association with nearsightedness, but a child reading a paper book at the same distance for the same hours is in a similar position. The bigger issue with screen time is what it replaces. A child indoors on a device is a child not outside in daylight, and outdoor time is the factor with the clearest protective signal.

Reading in dim light, another classic warning, causes temporary tiredness and headaches because the eye works harder to focus and the pupil dilates. It does not cause lasting damage or permanent nearsightedness, according to Harvard Health and other mainstream sources. The same applies to sitting close to a television: a child who sits very close may already be nearsighted and doing it because the screen is blurry from farther away, which reverses the cause and effect.

Digital eye strain, sometimes called computer vision syndrome, is real but different. It describes dry, tired, aching eyes after prolonged screen use, driven mostly by reduced blinking and sustained focusing. It is uncomfortable and it resolves with breaks. It is not a pathway to permanent vision change.

One habit that eye care professionals do encourage is the kind of break that involves looking into the distance, ideally by going outside. Framed that way, the advice is less about protecting eyes from screens and more about giving growing eyes the daylight and distance viewing they appear to need. That reframing, backed by the outdoor-time research, is more useful than blanket fear of devices.

Can kids outgrow myopia? What the following years usually look like

Parents ask this hopefully, and the answer needs to be gentle but clear: true myopia does not typically go away as a child grows. It generally does the opposite for a while.

According to Cleveland Clinic, nearsightedness is usually detected between the ages of six and fourteen. The NHS notes that it commonly begins around puberty and continues to progress until the eye stops growing, often in the late teens or around age twenty. During those years, a typical pattern is a small increase in prescription each year, faster in younger children and slowing as they approach adulthood. Children who become nearsighted earliest tend to end up with the highest final prescriptions simply because they have more years of growth ahead.

The confusion about “outgrowing” myopia often comes from two real phenomena. First, some very young children have a form of nearsightedness related to the eye’s natural development that does change as the eye matures, but this is uncommon and is identified by an eye care professional, not by waiting and hoping. Second, some children who squint or struggle with distance vision have a focusing problem rather than true myopia, and that can improve. Only an examination with dilating drops, which relax the focusing muscle so the true prescription can be measured, distinguishes these from structural nearsightedness.

So what do the following years actually look like for a family? Usually annual or more frequent exams, prescription updates as needed, and a conversation about whether progression is fast enough to warrant myopia control. The Mayo Clinic describes progression stabilizing in early adulthood for most people, though a minority continue to shift into their twenties.

A child cannot outgrow the eye length already gained. The goal of modern care is to limit how much more is added before growth stops.

Who is usually offered myopia control for children, and who is asked to wait

Myopia control means using lenses or eye drops designed to slow the rate at which a child’s nearsightedness increases. It is different from simply correcting vision, and it is not for every child. Eye care professionals weigh several factors, and the decision always rests with the treating team and family.

Children commonly considered for myopia control share some features:

• Onset at a young age, since earlier onset predicts more total progression.
• A prescription that has increased noticeably between exams, indicating active growth.
• One or two nearsighted parents, especially parents with high myopia.
• A lifestyle heavy in near work with limited outdoor time.

Children who are often asked to wait or simply monitored include those whose prescription has been stable across visits, those whose myopia is very mild and not changing, and teenagers nearing the age when progression typically slows on its own. Adults are generally not offered myopia control, because the eye has finished growing and the interventions target a process that is no longer active.

Some children are not candidates for particular options for practical reasons. Contact lens-based approaches require a child mature enough to handle lenses hygienically, or a parent willing to manage insertion and removal. Eye drops require reliable nightly routines and tolerance of side effects such as light sensitivity.

An eye care professional will also rule out other explanations for changing vision before attributing everything to ordinary myopia. That is why the first appointment is often mostly measurement and conversation rather than a treatment decision.

Families sometimes feel they have missed a window if a child is already twelve or thirteen. Progression can still be meaningful in early adolescence, so the question is worth asking at any age during childhood. The treating team can judge whether the expected benefit justifies the commitment.

What are the options for slowing myopia progression? A neutral comparison

Several approaches have evidence for slowing childhood myopia. None stops it entirely, none reverses eye length already gained, and each carries trade-offs. The table summarizes what they involve in neutral terms. Whether any is suitable for a particular child is a decision for the treating eye care professional.

Approach	What it involves	Proposed mechanism	Practical considerations
Standard single-vision glasses	Ordinary lenses that correct blur	None for progression; correction only	Safe and effective for clear vision; does not slow growth
Specialty myopia-control spectacle lenses	Glasses with a central clear zone and a peripheral treatment zone	Alters how light focuses on the peripheral retina to signal slower growth	Similar to wearing regular glasses; requires consistent daily wear
Multifocal or dual-focus soft contact lenses	Daily contact lenses with alternating focus zones	Peripheral defocus, as above	Requires lens hygiene and maturity; carries the usual contact lens infection risk
Orthokeratology	Rigid lenses worn overnight that temporarily reshape the cornea	Reshaped cornea creates peripheral defocus during the day	Glasses-free daytime; strict hygiene needed because of infection risk
Low-concentration atropine eye drops	A prescription drop, typically used nightly	Thought to act on receptors in the eye wall that regulate growth	Possible light sensitivity and near blur; does not correct vision, so glasses are still worn
Increased outdoor time	More daylight exposure, especially in early childhood	Bright light and distance viewing appear to restrain eye growth	Strongest evidence for preventing onset; free and low risk

Two honest caveats belong beside this table. First, most trials run two to three years, so long-term outcomes and what happens after stopping are still being studied. Second, individual response varies widely, and no eye care professional can promise a specific result for a specific child. The reasonable framing is that these approaches shift the odds, not that they guarantee an outcome.

How do atropine eye drops for myopia work, and what is the typical timeline?

Of all the myopia control options, eye drops attract the most questions, partly because a medicine feels more serious than a lens.

Atropine is a long-established drug that, at full strength, dilates the pupil and paralyzes the eye’s focusing muscle; it has been used for decades in eye examinations. Decades ago, clinicians noticed that children receiving it for other reasons progressed more slowly in their nearsightedness. The interest today is in much weaker preparations intended to slow eye growth while minimizing the pupil and focusing effects.

How it slows growth is not fully understood. The drug blocks a family of receptors called muscarinic receptors, and these exist not only in the focusing muscle but also in the retina and the sclera, the white outer wall of the eye. The leading hypothesis is that acting on those receptors in the eye wall changes the biochemical signals that tell the eyeball to lengthen. The focusing effect on the muscle, once assumed to be the mechanism, appears to be largely incidental.

Typical timelines, as described in published trials, involve nightly use over at least two years, with prescription and eye length checked every six to twelve months to judge response. Some children respond well, others modestly, and a proportion show little change. A rebound in progression after stopping has been reported, particularly with higher concentrations, so how and when to stop is itself a clinical decision.

Common side effects include glare and light sensitivity from a slightly larger pupil and some difficulty focusing up close, which are more pronounced at higher concentrations. The drops do not correct vision, so children continue wearing glasses.

Whether to start, which preparation is appropriate, and how long to continue are decisions for the prescribing eye care professional, who will weigh the child’s rate of progression, age, and tolerance. Nothing here should be read as a recommendation to start or change any medicine.

Which ethnicity has the most myopia? What the global data really shows

The question is asked often and deserves a careful answer, because the way it is usually framed leads to the wrong conclusion.

A widely cited analysis in the journal Ophthalmology pooled population studies worldwide and estimated that nearsightedness affected roughly 23 percent of the global population at the turn of the century, with a projection of about half the world’s population by mid-century if trends continue. High myopia was projected to affect nearly one in ten people by that point. The same analysis found the highest prevalence in East and Southeast Asia, with studies in some urban areas reporting that the large majority of young adults were nearsighted.

It would be easy to read that as “East Asian people are genetically prone to myopia.” The evidence points elsewhere. Prevalence in those regions was far lower a few generations ago, and it rose in step with intensive schooling, urbanization, and reduced outdoor time. Children of East Asian heritage raised in environments with more outdoor activity show lower rates than their peers in dense urban school systems. Meanwhile, prevalence has also climbed sharply in Europe and North America, suggesting the environment, not ancestry, is doing most of the work.

Genetics does contribute to individual risk, and there may be modest differences in susceptibility between populations. But the dramatic regional gaps are best explained by how children spend their days.

The World Health Organization estimates that more than two billion people worldwide have some form of vision impairment, and that unaddressed refractive error, including uncorrected nearsightedness, remains one of the leading causes. That framing matters more than any ethnicity ranking: the global challenge is less about who gets myopia and more about ensuring every child who needs correction receives it and that progression is monitored where care is available.

Is myopia linked to autism or other health conditions?

This question circulates online, often with more confidence than the evidence supports.

There is no established causal link between nearsightedness and autism. Some studies have reported that refractive errors of various kinds, including farsightedness, astigmatism, and nearsightedness, appear somewhat more often in children with autism and other neurodevelopmental conditions than in the general population. The reasons are not clear and may include shared developmental factors, differences in visual behavior, or simply that vision problems are detected differently in children who find standard eye tests difficult. The evidence is limited, mixed, and does not suggest that myopia causes autism or that autism causes myopia.

The practical takeaway is more useful than the statistical one. Children with autism or other developmental differences may not report blurry vision, may find eye charts challenging, and may be assumed to be inattentive when they are actually struggling to see. Eye care professionals experienced with children have techniques for assessing vision without relying on verbal responses, so a full eye examination is worth arranging even when a child cannot complete a school screening.

Myopia does have well-documented associations with certain other conditions. Premature birth and low birth weight raise the risk of nearsightedness. Some connective tissue disorders, such as Marfan syndrome, are associated with high myopia because they affect the structure of the eye wall. Certain inherited retinal conditions also present with early, severe nearsightedness. When myopia appears very early, progresses unusually fast, or reaches high levels, an eye care professional may look for one of these underlying causes.

For the overwhelming majority of nearsighted children, though, myopia is an isolated finding related to eye growth, not a marker of another condition. Treating it as a sign of something hidden is one of the myopia myths that adds anxiety without adding information.

Is high myopia dangerous? Why the total amount matters later in life

Here is where a myth works in the opposite direction. Many people believe nearsightedness is a trivial inconvenience, fully solved by glasses. For mild myopia, that is close to true. For high myopia, it understates a genuine long-term concern.

Recall the stretched balloon. As the eyeball lengthens, the retina and the layers beneath it are pulled thinner. Over decades, that stretching raises the risk of several conditions, according to the Mayo Clinic and the National Eye Institute:

• Retinal detachment, where the retina pulls away from the back of the eye, a sight-threatening emergency.
• Myopic macular degeneration, damage to the central retina caused by stretching rather than age.
• Glaucoma, damage to the optic nerve often associated with eye pressure.
• Cataracts, which tend to develop earlier in highly nearsighted eyes.

These risks rise with the degree of myopia. A person with two diopters of nearsightedness has only a slightly elevated risk compared with someone with normal vision. A person with eight diopters faces a substantially higher lifetime risk, and the risk does not disappear after laser vision correction, because surgery reshapes the cornea without shortening the eye.

This is the real reason eye care professionals now talk about slowing progression in children. The goal is not to spare a teenager a slightly thicker lens. It is to reduce the total eye length a person carries into their fifties and sixties, when the stretched retina becomes vulnerable. Even a modest reduction in final prescription is thought to lower lifetime risk of these complications, though the long-term data confirming that benefit are still accumulating.

People with high myopia at any age benefit from regular dilated eye examinations, where the retina can be inspected directly. The blur can be fixed with a lens. The retina needs watching.

Nearsightedness myths: what people often get wrong

Some of the persistent beliefs about nearsightedness, held up against what the evidence actually shows.

“Wearing glasses full time makes your eyes dependent on them.” Vision does not become dependent on lenses. What changes is perception: once a person experiences clear vision, the old blur feels worse by comparison. The eye itself has not weakened.

“Eye exercises can reverse myopia.” No exercise program has been shown to shorten the eyeball or reduce true nearsightedness. Exercises may ease focusing fatigue, which is a different problem. Programs promising to eliminate the need for glasses are not supported by evidence.

“Carrots or supplements will fix it.” Vitamin A deficiency causes night blindness and other problems, but supplementing a child who eats an ordinary diet does not affect eye length. No nutrient has been shown to prevent or slow myopia.

“Laser surgery treats the underlying condition.” Refractive surgery reshapes the cornea so light focuses correctly. It corrects blur but leaves the elongated eye, and its associated retinal risks, unchanged. It is also generally not performed until the prescription has been stable, usually in adulthood.

“If a child squints, they need glasses immediately.” Squinting can indicate nearsightedness, but it can also reflect farsightedness, astigmatism, light sensitivity, or a habit. Only an eye examination determines the cause.

“Myopia is just about needing glasses.” As the previous section explained, high myopia carries meaningful long-term risks to the retina. Treating it as purely cosmetic is the myth most likely to cause harm later.

“Nothing can be done except updating the prescription.” This was largely true a generation ago. Today, evidence-based options exist to slow progression in children, and outdoor time appears to reduce onset. The conversation with an eye care professional has more to offer than it once did.

Questions to ask your eye care team about a child's myopia

A good appointment is a conversation, and parents who arrive with questions tend to leave with clearer plans. Some worth raising:

• How much has the prescription changed since the last exam, and is that faster or slower than typical for this age?
• Was the measurement taken with dilating drops, and if not, would that give a more accurate picture?
• Is my child a candidate for myopia control, and if not, what would change that assessment?
• Which options are realistic for our family given my child’s age, maturity, and daily routine?
• What side effects or practical difficulties should we expect with each option?
• How will we know whether an approach is working, and how often will we check?
• What happens when we eventually stop, and how is that decision made?
• How much outdoor time would you suggest, and how can we build it into an ordinary school week?
• Are there any signs of something other than ordinary myopia that you are watching for?
• How often should my child have a full eye examination from here on?

Adults with established myopia have their own set of useful questions, particularly if the prescription is high:

• How thin does my retina look, and are there any areas that need monitoring?
• What symptoms would mean I should be seen urgently?
• Does my level of myopia change how often I should have a dilated examination?
• If I am considering refractive surgery, is my prescription stable enough, and what would surgery not change about my eye?

It is also reasonable to ask an eye care professional to explain the reasoning behind any recommendation in plain language. Myopia care involves trade-offs between benefit, side effects, cost of effort, and uncertainty, and families make better decisions when those trade-offs are spelled out. The decision itself always sits with the treating team and the patient or family together.

When to call your doctor about nearsightedness: red-flag signs

Most nearsightedness is managed at routine appointments, and the NHS suggests eye tests roughly every two years for adults, with more frequent checks for children whose vision is changing or as advised. Some situations should not wait for the next scheduled visit.

Seek same-day or emergency eye care for any of the following, particularly if you or your child has moderate or high myopia:

• A sudden shower of new floaters, the small specks or threads that drift across vision.
• Flashes of light in the side of vision, like a camera flash or lightning, especially when repeated.
• A shadow, curtain, or dark area moving across part of the visual field.
• Sudden loss or marked blurring of vision in one eye that does not clear with blinking.
• Straight lines that suddenly appear wavy or distorted, or a blank patch in central vision.
• Eye pain with redness, halos around lights, nausea, or a cloudy cornea.

These can indicate retinal detachment, a retinal tear, bleeding at the macula, or acutely raised eye pressure. Prompt treatment matters for preserving sight, so err on the side of being seen.

Arrange a prompt but non-emergency appointment for:

• A noticeable change in a child’s distance vision between exams, or new squinting, head tilting, or sitting very close to screens.
• A rapid prescription change in an adult, which can signal something other than ordinary myopia.
• Persistent headaches or eye discomfort after reading or screen use.
• Redness, pain, discharge, or light sensitivity in anyone using contact lenses, including overnight lenses, since infection can progress quickly.
• Any concern that a child cannot see well but cannot describe it, particularly children with developmental differences.

When in doubt, call. Eye care professionals would far rather evaluate a false alarm than see a retinal detachment that has been developing for a week. That instinct to check early is the one habit that genuinely protects nearsighted eyes over a lifetime.

Frequently asked questions

Can myopia be reversed once it has developed?

Not with any method supported by evidence. Nearsightedness reflects an eyeball that has grown too long, and no exercise, diet, drop, or lens shortens it. Glasses, contact lenses and refractive surgery correct the resulting blur, and myopia control approaches in children can slow further growth, but the eye length already gained remains. Programs claiming to reverse myopia naturally are not backed by clinical evidence.

Does myopia get worse with age in adults?

Usually not in the way it does in childhood. Progression typically slows and stabilizes in the late teens or early twenties once the eye stops growing. Some adults see small shifts related to intensive near work, pregnancy, certain medicines, or early cataract changes. A sudden or large prescription change in adulthood is worth a full eye examination rather than assuming ordinary myopia.

Do glasses make myopia worse if a child wears them all day?

No. Full-time wear of correctly prescribed glasses does not accelerate nearsightedness. The prescription rises because the eye is still growing, and it would rise whether or not glasses were worn. Research on deliberately under-correcting children found progression was not slowed and in some studies was faster, so leaving a child slightly blurry offers no protection.

Can kids outgrow myopia on their own?

True structural myopia does not go away with age; it typically increases until the eye stops growing. Some very young children have developmental nearsightedness that changes as the eye matures, and some children have focusing problems rather than true myopia, which can improve. Only an eye examination, usually with dilating drops, distinguishes these situations from ordinary progressive nearsightedness.

Which ethnicity has the most myopia?

Population studies report the highest prevalence in East and Southeast Asia, where a large majority of young adults in some urban areas are nearsighted. However, rates there were far lower a few generations ago and rose alongside intensive schooling and reduced outdoor time, and prevalence is also climbing in Europe and North America. Environment, not ancestry, appears to explain most of the regional difference.

Is myopia linked to autism?

There is no established causal link. Some studies report that refractive errors of all types appear somewhat more often in children with autism and other neurodevelopmental conditions, but the evidence is limited and the reasons are unclear. The practical point is that children who find standard eye tests difficult still benefit from a full examination by a professional experienced in assessing vision without verbal responses.

What are the main theories behind myopia?

The leading explanations are heredity, near work, insufficient outdoor light exposure, and peripheral defocus, the idea that how light focuses on the edges of the retina guides eye growth. Genetics sets a child’s susceptibility, but the rapid rise in prevalence points to environmental triggers, with reduced daylight exposure having the strongest recent support. Most researchers view myopia as a gene-environment condition rather than the result of any single cause.

How does outdoor time help with myopia control for children?

Children who spend more time outdoors are less likely to become nearsighted, and school programs that added outdoor periods reduced new cases. The leading hypothesis is that bright daylight stimulates dopamine release in the retina, which appears to restrain eye lengthening. Distance viewing outdoors may also contribute. The evidence is strongest for preventing onset rather than slowing established myopia.

Does laser eye surgery fix nearsightedness permanently?

Refractive surgery reshapes the cornea so light focuses on the retina, which corrects blur for most suitable adults with stable prescriptions. It does not shorten the eyeball, so the long-term retinal risks associated with high myopia remain unchanged after surgery. Whether someone is a suitable candidate, and what the procedure can and cannot change, is a discussion for an eye surgeon.

Why do eye doctors care about slowing myopia if glasses fix it?

Because the total amount of nearsightedness a person accumulates affects lifetime risk of retinal detachment, myopic macular degeneration, glaucoma and early cataract. These risks rise with eye length, so limiting how much a child’s eye lengthens before growth stops is thought to lower risk decades later. Glasses correct the blur; they do not address the stretched retina underneath.

References

• NHS – Short-sightedness (myopia)
• Cleveland Clinic – Myopia (Nearsightedness)
• National Eye Institute (NIH) – Nearsightedness (Myopia)
• World Health Organization – Blindness and vision impairment fact sheet

This article is for general information only and is not a substitute for professional medical advice. Please consult a qualified doctor about your individual situation.