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Rickets: What Patients Need to Know

9 min read Published July 18, 2026
Healthcare professionals and patients in a hospital corridor.
Quick answer

Rickets affects developing bones in children and can lead to bone pain, delayed growth, and bowed legs. The most common cause is vitamin D deficiency, but low calcium, phosphate problems, and certain inherited conditions can also be involved.

Key Takeaways

  • Rickets affects developing bones in children and can lead to bone pain, delayed growth, and bowed legs.
  • The most common cause is vitamin D deficiency, but low calcium, phosphate problems, and certain inherited conditions can also be involved.
  • Diagnosis usually includes a physical exam, blood tests, and sometimes X-rays to assess bone changes.
  • Treatment depends on the cause and may include vitamin D, calcium, phosphate management, and treatment of underlying medical conditions.
  • Children with signs of bone deformity, delayed growth, or unexplained muscle weakness should be evaluated by a doctor.

Medically reviewed by the Acıbadem International Medical Board — July 18, 2026

Dr. Bahadır Kaynarkaya, MD Dr. Mohamed Al-Qadi, MD Dr. Şule Eren, MD Dr. Tarek Arafat, MD

Rickets is a condition in children in which growing bones become soft and weak, most often because the body does not have enough vitamin D, calcium, or phosphate. With timely diagnosis and treatment, many children improve well and can support healthier bone growth.

Overview

Rickets is a disorder that affects children’s growing bones. It happens when bones do not mineralize properly, which means they do not become as strong and hard as they should. As a result, bones may become soft, weak, or misshapen during growth.

In many cases, rickets is linked to vitamin D deficiency, but it is not the only cause. Some children develop rickets because of low calcium intake, problems absorbing nutrients from the intestine, kidney disorders, or inherited conditions that affect how the body handles phosphate. Understanding the underlying cause is important because treatment is directed at the reason the bones are weakening.

Rickets is different from osteomalacia, which describes similar bone softening in adults after growth plates have closed. In children, the presence of active growth plates means the condition can affect height, leg shape, walking, and dental development. Early medical care can help reduce the risk of lasting bone deformities and support healthier growth.

Symptoms and how rickets may appear

Pediatric doctor consulting a young patient in hospital bed with medical equipment.

The signs of rickets can develop gradually and may be easy to miss at first. Some children seem tired, irritable, or less physically active than usual. Others complain of bone pain or tenderness, especially in the legs, pelvis, spine, or ribs. Muscle weakness can also make a child seem less steady when standing or walking.

As the condition progresses, changes in the shape of growing bones may become more noticeable. Bowed legs are a well-known sign, but children may also develop knock knees, thickened wrists or ankles, delayed closure of the soft spots of the skull, or changes in the chest wall. Some children have delayed growth or a shorter height than expected for age.

Dental issues can also occur. Teeth may erupt later than expected, enamel may be weaker, and the risk of cavities may be higher. In infants and younger children, severe mineral imbalance can occasionally lead to muscle cramps or seizures related to low calcium, which requires urgent medical attention.

  • Bone pain or tenderness
  • Delayed growth
  • Bowed legs or other limb deformities
  • Muscle weakness
  • Delayed motor development
  • Dental problems

Causes and risk factors

Pediatric consultation at Acibadem Hospital with doctor and young patient.

The most common cause of rickets worldwide is not getting enough vitamin D. Vitamin D helps the body absorb calcium and phosphate, which are both essential for strong bones. Children may become deficient if they have limited sun exposure, low dietary intake, or conditions that reduce absorption from the digestive tract.

Low calcium intake can also contribute, even when vitamin D levels are not severely reduced. In other children, the problem is phosphate handling. This may be due to kidney disorders or inherited conditions such as phosphate-wasting disorders. In these cases, standard vitamin D alone may not be enough, and more specialized treatment is needed.

Risk factors include exclusive breastfeeding without appropriate vitamin D supplementation when recommended, darker skin pigmentation, limited outdoor exposure, malabsorption disorders, chronic liver or kidney disease, and certain medications that affect vitamin D metabolism. Children with digestive conditions such as celiac disease may be at higher risk because nutrient absorption can be impaired. A child with an unusual or severe presentation may need evaluation for a rare inherited form of rickets.

Because several medical issues can lead to weak bones, doctors may also consider other causes of bone fragility. In some situations, the evaluation overlaps with the assessment of bone weakness disorders or metabolic bone disease more broadly, especially when symptoms are not typical.

How doctors diagnose rickets

Diagnosis starts with a careful medical history and physical examination. A doctor asks about diet, vitamin use, growth, sun exposure, family history, and any symptoms such as pain, weakness, delayed walking, or changes in leg shape. On examination, the doctor looks for bone tenderness, deformities, growth delay, and dental or muscle findings.

Blood tests are usually important because they help show how the body is handling bone minerals. Tests may include vitamin D, calcium, phosphate, alkaline phosphatase, and parathyroid hormone. Depending on the situation, kidney and liver function tests or urine studies may also be needed. These results help distinguish nutritional rickets from inherited or kidney-related causes.

X-rays can show characteristic changes at the growth plates and in the long bones, especially around the wrists and knees. Imaging helps confirm the diagnosis and assess severity. If the pattern is complex, doctors may use MRI scanning or other tests to evaluate related concerns, though MRI is not usually the first test for straightforward rickets.

When symptoms suggest a broader endocrine or metabolic issue, referral to specialists may be appropriate. In some children, assessment may also include pediatric endocrinology input, particularly when growth is affected or when laboratory findings do not fit simple vitamin D deficiency.

Treatment options and long-term outlook

Treatment depends on the cause. For nutritional rickets, the main goals are to correct vitamin D deficiency, improve calcium intake, and monitor bone recovery. Doctors usually recommend vitamin D and, when needed, calcium supplementation. Families should follow the prescribed plan carefully, because treatment needs vary by age, severity, and laboratory results.

If rickets is caused by phosphate loss, kidney disease, or an inherited condition, treatment can be more specialized. A child may need phosphate supplements, active forms of vitamin D, or treatment of the underlying medical problem. Conditions related to kidney function may involve coordinated care through nephrology services.

Children are usually monitored over time with repeat blood tests, growth checks, and sometimes follow-up X-rays. Many improve well when treatment starts early. Bone pain often lessens, blood test abnormalities may normalize, and growth can continue more normally. However, established bone deformities may take time to improve and some may persist if the diagnosis is delayed.

In a smaller number of cases, significant bone deformity may require orthopedic assessment. Surgery is not needed for most children with nutritional rickets, but it can be considered if leg alignment problems remain after the metabolic problem has been corrected and the child continues to grow.

Prevention and self-care for families

Prevention focuses on supporting healthy bone mineralization during growth. A balanced diet that provides enough vitamin D, calcium, and other nutrients is important. Depending on a child’s age and feeding pattern, a doctor may recommend vitamin D supplementation, especially in infancy or when dietary intake is limited.

Safe sun exposure can help the body make vitamin D, but recommendations vary according to skin type, climate, season, and sun safety concerns. Because too much sun exposure has risks, families should not rely on sunlight alone without discussing prevention with a healthcare professional. Food sources such as fortified dairy or plant-based products, eggs, and oily fish may also help, depending on the child’s diet.

Children with chronic digestive, liver, or kidney conditions may need more careful monitoring. Families should keep regular appointments if a child has a disorder that affects growth or nutrient absorption. When rickets is suspected or confirmed, giving supplements exactly as advised is important, because too little may not work and too much can also be harmful.

It can also help to discuss the child’s full health picture with the care team, including any medications, feeding difficulties, developmental concerns, or special diets. For international patients who need coordinated evaluation, Acibadem International’s multidisciplinary specialists in JCI-accredited hospitals diagnose and treat rickets and related bone and metabolic conditions.

When to seek medical care

Medical evaluation is important if a child has bowed legs, delayed growth, persistent bone pain, unexplained muscle weakness, or delayed motor milestones such as standing or walking. Parents should also seek advice if teeth are erupting very late, if the child fractures bones easily, or if there is a strong family history of bone or mineral disorders.

Urgent care is needed if a child has muscle spasms, seizures, severe weakness, or signs of very low calcium. These symptoms can have several causes, but they should be assessed promptly. A doctor can determine whether rickets, a mineral imbalance, or another condition is responsible.

It is also reasonable to consult a doctor if a child has risk factors for nutrient deficiency, chronic digestive disease, kidney disease, or poor growth even without obvious leg deformity. Early assessment often allows simpler treatment and helps protect normal bone development.

Frequently asked questions

Is rickets the same as vitamin D deficiency?

Not exactly. Vitamin D deficiency is a common cause of rickets, but rickets can also result from low calcium, phosphate problems, kidney disease, or inherited disorders. The term rickets refers to the effect these problems have on growing bones.

Can adults get rickets?

Adults do not get rickets because their growth plates have already closed. However, adults can develop osteomalacia, a related condition in which bones become soft due to poor mineralization. The causes may overlap, especially vitamin D deficiency.

Can rickets be cured?

Many cases of nutritional rickets improve very well when the cause is identified and treated early. Vitamin D and calcium correction often leads to better bone health and symptom improvement. Some bone deformities may take time to improve, and severe longstanding changes may not fully reverse.

What foods help prevent rickets?

Foods that provide vitamin D and calcium can support healthy bones. Depending on the diet, this may include fortified milk or fortified alternatives, dairy products, eggs, and certain fish. A doctor or dietitian can help if a child has allergies, a restricted diet, or feeding difficulties.

Does every child with bowed legs have rickets?

No. Mild bowing can be part of normal development in some young children, and other bone or growth conditions can also affect leg shape. A doctor can decide whether the finding is normal for age or whether testing is needed.

How long does treatment for rickets take?

Treatment length depends on the cause, severity, and how the child responds. Some children improve within weeks to months, but monitoring often continues longer to confirm normal mineral levels and healthy growth. Children with inherited or kidney-related forms may need ongoing care.

This article is for general information only and is not a substitute for professional medical advice. Please consult a qualified doctor about your individual situation.

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Dr. Bahadır Kaynarkaya
Dr. Bahadır Kaynarkaya, MD
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